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Cognitive deficits in a mouse model of pre-manifest Parkinson's disease

Cognitive deficits in a mouse model of pre-manifest Parkinson's disease

http://www.wikidata.org/entity/Q37669038

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Disease-modifying therapeutic directions for Lewy-Body dementias A progressive mouse model of Parkinson's disease: the Thy1-aSyn ("Line 61") mice Characterization of cognitive deficits in rats overexpressing human alpha-synuclein in the ventral tegmental area and medial septum using recombinant adeno-associated viral vectors Toxin-Induced Experimental Models of Learning and Memory Impairment Late-stage α-synuclein accumulation in TNWT-61 mouse model of Parkinson's disease detected by diffusion kurtosis imaging.The neurobiological basis of cognitive impairment in Parkinson's disease.Novel AAV-based rat model of forebrain synucleinopathy shows extensive pathologies and progressive loss of cholinergic interneurons Soluble α-synuclein is a novel modulator of Alzheimer's disease pathophysiology.Network dysfunction in α-synuclein transgenic mice and human Lewy body dementia.α-Synuclein interferes with the ESCRT-III complex contributing to the pathogenesis of Lewy body disease.Fasudil attenuates aggregation of α-synuclein in models of Parkinson's disease Rodent models of Parkinson's disease: beyond the motor symptomatology A Molecular Tweezer Ameliorates Motor Deficits in Mice Overexpressing α-Synuclein.Human LRRK2 G2019S mutation represses post-synaptic protein PSD95 and causes cognitive impairment in transgenic mice Basal Forebrain Cholinergic Circuits and Signaling in Cognition and Cognitive Decline.Non-motor features of Parkinson disease.α-synuclein conformational antibodies fused to penetratin are effective in models of Lewy body disease.Intracellular soluble α-synuclein oligomers reduce pyramidal cell excitability.Impaired baroreflex function in mice overexpressing alpha-synuclein.The Cholinergic System Modulates Memory and Hippocampal Plasticity via Its Interactions with Non-Neuronal Cells.Social Cognition Impairments in Mice Overexpressing Alpha-Synuclein Under the Thy1 Promoter, a Model of Pre-manifest Parkinson's Disease.α-synuclein interacts with PrPC to induce cognitive impairment through mGluR5 and NMDAR2B.A Protein Aggregation Inhibitor, Leuco-Methylthioninium Bis(Hydromethanesulfonate), Decreases α-Synuclein Inclusions in a Transgenic Mouse Model of Synucleinopathy.Sleep dysfunction and EEG alterations in mice overexpressing alpha-synuclein.Locomotor differences in mice expressing wild-type human α-synuclein.

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Cognitive deficits in a mouse model of pre-manifest Parkinson's disease

http://www.wikidata.org/entity/Q37669038

description

article científic

@ca

article scientifique

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articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 22 February 2012

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Cognitive deficits in a mouse model of pre-manifest Parkinson's disease

@en

Cognitive deficits in a mouse model of pre-manifest Parkinson's disease.

@nl

type

label

Cognitive deficits in a mouse model of pre-manifest Parkinson's disease

@en

Cognitive deficits in a mouse model of pre-manifest Parkinson's disease.

@nl

prefLabel

Cognitive deficits in a mouse model of pre-manifest Parkinson's disease

@en

Cognitive deficits in a mouse model of pre-manifest Parkinson's disease.

@nl

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J.1460-9568.2012.08012.X

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European Journal of Neuroscience

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Cognitive deficits in a mouse model of pre-manifest Parkinson's disease

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Chunni Zhu

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Diana Dinh

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Eddie C Garcia

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Eileen Ruth Torres

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Iddo Magen

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Katherine M Cardiff

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Krystal De La Rosa

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Maria Sanchez

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Sheila M Fleming

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P2860

Mutation in the alpha-synuclein gene identified in families with Parkinson's disease

Staging of brain pathology related to sporadic Parkinson's disease

Cognitive function in early Parkinson's disease: a population-based study

Obstacle crossing among people with Parkinson disease is influenced by concurrent music.

Up-regulating BDNF with an ampakine rescues synaptic plasticity and memory in Huntington's disease knockin mice.

L-dopa medication in Parkinson's disease restores activity in the motor cortico-striatal loop but does not modify the cognitive network

Enhanced frontal function in Parkinson's disease

A flexible sequential learning deficit in patients with Parkinson's disease: a 2 x 8 button-press task.

Executive impairment in Parkinson's disease: response automaticity and task switching.

{alpha}-Synuclein gene duplication impairs reward learning.

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870-882

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scholarly article

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10.1111/J.1460-9568.2012.08012.X

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6

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35

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Eliezer Masliah

J. David Jentsch

Marie-Françoise Chesselet

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2012-02-22T00:00:00Z

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22356593

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Parkinson's disease

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3967873

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