Host immune response to rapidly growing mycobacteria, an emerging cause of chronic lung disease.
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US Cystic Fibrosis Foundation and European Cystic Fibrosis Society consensus recommendations for the management of non-tuberculous mycobacteria in individuals with cystic fibrosisAn Update on Global Tuberculosis (TB)Acetic Acid, the active component of vinegar, is an effective tuberculocidal disinfectantIncreased virulence of an epidemic strain of Mycobacterium massiliense in miceHost response to nontuberculous mycobacterial infections of current clinical importance.Importance of reciprocal balance of T cell immunity in Mycobacterium abscessus complex lung disease.Differential immune responses to Segniliparus rotundus and Segniliparus rugosus infection and analysis of their comparative virulence profiles.Complete genome sequence and comparative genomic analysis of Mycobacterium massiliense JCM 15300 in the Mycobacterium abscessus group reveal a conserved genomic island MmGI-1 related to putative lipid metabolismInduction of heme oxygenase-1 contributes to survival of Mycobacterium abscessus in human macrophages-like THP-1 cells.Essential engagement of Toll-like receptor 2 in initiation of early protective Th1 response against rough variants of Mycobacterium abscessus.Structure elucidation and biological activity of antibacterial compound from Micromonospora auratinigra, a soil Actinomycetes.Mycobacterium abscessus activates the NLRP3 inflammasome via Dectin-1-Syk and p62/SQSTM1.Susceptibility of Mycobacterium abscessus to antimycobacterial drugs in preclinical modelsMolecular Identification of Clinical Isolates of Mycobacterium fortuitum by Random Amplified Polymorphic DNA (RAPD) Polymerase Chain Reaction and ERIC PCR.New rapid scheme for distinguishing the subspecies of the Mycobacterium abscessus group and identifying Mycobacterium massiliense isolates with inducible clarithromycin resistanceMycobacterium massiliense induces inflammatory responses in macrophages through Toll-like receptor 2 and c-Jun N-terminal kinase.Mycobacterium fortuitum induces A20 expression that impairs macrophage inflammatory responses.Gallium Compounds Exhibit Potential as New Therapeutic Agents against Mycobacterium abscessus.Innate signaling mechanisms controlling Mycobacterium chelonae-mediated CCL2 and CCL5 expression in macrophages.Whole-genome sequencing to identify transmission of Mycobacterium abscessus between patients with cystic fibrosis: a retrospective cohort studyTreatment of Mycobacterium abscessus: all macrolides are equal, but perhaps some are more equal than othersDisseminated Mycobacterium abscessus infection and showerheads, Taiwan.Clinical significance of respiratory isolates for Mycobacterium abscessus complex from pediatric patients.The use of a two-gene sequencing approach to accurately distinguish between the species within the Mycobacterium abscessus complex and Mycobacterium chelonae.Causative species of nontuberculous mycobacterial lung disease and comparative investigation on clinical features of Mycobacterium abscessus complex disease: A retrospective analysis for two major hospitals in a subtropical region of Japan.Mycobacterial genotypes are associated with clinical manifestation and progression of lung disease caused by Mycobacterium abscessus and Mycobacterium massiliense.Epidemiology and Predictors of NTM Pulmonary Infection in Taiwan - a Retrospective, Five-Year Multicenter Study.galK-based suicide vector mediated allelic exchange in Mycobacterium abscessus.Mycobacterium abscessus Smooth And Rough Morpohotypes Form Antimicrobial-Tolerant Biofilm Phenotypes But Are Killed by Acetic Acid.Middle-aged to elderly women have a higher asymptomatic infection rate with Mycobacterium avium complex, regardless of body habitus.Anomalies in T Cell Function Are Associated With Individuals at Risk of Mycobacterium abscessus Complex Infection.Hydroalcoholic Extract and Ethyl Acetate Fraction of Leaves Decrease the Inflammatory Response to Subsp.
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Host immune response to rapidly growing mycobacteria, an emerging cause of chronic lung disease.
description
article científic
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article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 15 January 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Host immune response to rapidl ...... cause of chronic lung disease.
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Host immune response to rapidl ...... cause of chronic lung disease.
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type
label
Host immune response to rapidl ...... cause of chronic lung disease.
@en
Host immune response to rapidl ...... cause of chronic lung disease.
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prefLabel
Host immune response to rapidl ...... cause of chronic lung disease.
@en
Host immune response to rapidl ...... cause of chronic lung disease.
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Host immune response to rapidl ...... cause of chronic lung disease.
@en
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Edward D Chan
Ian M Orme
Marinka Kartalija
Xiyuan Bai
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P304
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10.1165/RCMB.2009-0276TR
P577
2010-01-15T00:00:00Z