T cell costimulation blockade blunts pressure overload-induced heart failure. - Wikidata - wikimedia - TriplyDB

T cell costimulation blockade blunts pressure overload-induced heart failure.

T cell costimulation blockade blunts pressure overload-induced heart failure.

http://www.wikidata.org/entity/Q37690286

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Unmet Needs in the Pathogenesis and Treatment of Cardiovascular Comorbidities in Chronic Inflammatory Diseases.Myocardial Metabolism Under Control of a Cytokine Receptor.T helper cells with specificity for an antigen in cardiomyocytes promote pressure overload-induced progression from hypertrophy to heart failure.Regnase-1 and Roquin Nonredundantly Regulate Th1 Differentiation Causing Cardiac Inflammation and Fibrosis.T cell responses to tumor: how dominant assumptions on immune activity led to a neglect of pathological functions, and how evolutionary considerations can help identify testable hypotheses for improving immunotherapy.Immunotherapy for cardiovascular disease.Caspase-1 regulate AngII-induced cardiomyocyte hypertrophy via upregulation of IL-1β.Immune mechanisms in heart failure.T-cell costimulation blockade is effective in experimental digestive and lung tissue fibrosis Ectopic overexpression of Kir6.1 in the mouse heart impacts on the life expectancy

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T cell costimulation blockade blunts pressure overload-induced heart failure.

http://www.wikidata.org/entity/Q37690286

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 06 March 2017

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

T cell costimulation blockade blunts pressure overload-induced heart failure.

@en

T cell costimulation blockade blunts pressure overload-induced heart failure.

@nl

type

label

T cell costimulation blockade blunts pressure overload-induced heart failure.

@en

T cell costimulation blockade blunts pressure overload-induced heart failure.

@nl

prefLabel

T cell costimulation blockade blunts pressure overload-induced heart failure.

@en

T cell costimulation blockade blunts pressure overload-induced heart failure.

@nl

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Nature Communications

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T cell costimulation blockade blunts pressure overload-induced heart failure.

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Andrea Fumero

http://www.w3.org/2001/XMLSchema#string

Anne Marie Ormbostad Berre

http://www.w3.org/2001/XMLSchema#string

Claudia Sardi

http://www.w3.org/2001/XMLSchema#string

Cristiano Rumio

http://www.w3.org/2001/XMLSchema#string

Debora Vignali

http://www.w3.org/2001/XMLSchema#string

Elisa Di Pasquale

http://www.w3.org/2001/XMLSchema#string

Elisa Martini

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Federica Riva

http://www.w3.org/2001/XMLSchema#string

Giuliana Roselli

http://www.w3.org/2001/XMLSchema#string

Roberto Papait

http://www.w3.org/2001/XMLSchema#string

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Left Ventricular T-Cell Recruitment Contributes to the Pathogenesis of Heart Failure.

Regulatory T cells exert checks and balances on self tolerance and autoimmunity

The chemokine system in diverse forms of macrophage activation and polarization

Cytokine storm in a phase 1 trial of the anti-CD28 monoclonal antibody TGN1412

Intermittent pressure overload triggers hypertrophy-independent cardiac dysfunction and vascular rarefaction.

The epidemiology of heart failure, based on data for 2.1 million inhabitants in Sweden.

Differential contribution of monocytes to heart macrophages in steady-state and after myocardial infarction.

Interleukin 6 mediates myocardial fibrosis, concentric hypertrophy, and diastolic dysfunction in rats.

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14680

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scholarly article

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10.1038/NCOMMS14680

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8

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Gianluigi Condorelli

Carolina M Greco

Giuseppe Faggian

Daniele Catalucci

Pierluigi Carullo

Marinos Kallikourdis

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2017-03-06T00:00:00Z

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1084128908

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