Direct repression of MYB by ZEB1 suppresses proliferation and epithelial gene expression during epithelial-to-mesenchymal transition of breast cancer cells.
about
The miRacle in Pancreatic Cancer by miRNAs: Tiny Angels or Devils in Disease ProgressionMinimal residual disease in breast cancer: an overview of circulating and disseminated tumour cellsHypoxia-inducible factor 1 and breast cancer metastasisBGRMI: A method for inferring gene regulatory networks from time-course gene expression data and its application in breast cancer researchTransitions between epithelial and mesenchymal states during cell fate conversions.Epithelial-to-mesenchymal transition is dispensable for metastasis but induces chemoresistance in pancreatic cancer.Stimulus-dependent differences in signalling regulate epithelial-mesenchymal plasticity and change the effects of drugs in breast cancer cell lines.Deciphering Seed Sequence Based Off-Target Effects in a Large-Scale RNAi Reporter Screen for E-Cadherin ExpressionCDK9 inhibitors selectively target estrogen receptor-positive breast cancer cells through combined inhibition of MYB and MCL-1 expression.Circulating tumor cells and epithelial, mesenchymal and stemness markers: characterization of cell subpopulations.Epithelial Mesenchymal Transition: a double-edged sword.New Insights on COX-2 in Chronic Inflammation Driving Breast Cancer Growth and Metastasis.Oxidative stress shapes breast cancer phenotype through chronic activation of ATM-dependent signaling.The hypoxic microenvironment: A determinant of cancer stem cell evolution.The presence of androgen receptor elements regulates ZEB1 expression in the absence of androgen receptor.Roles of Grainyhead-like transcription factors in cancer.Epithelial requirement for in vitro proliferation and xenograft growth and metastasis of MDA-MB-468 human breast cancer cells: oncogenic rather than tumor-suppressive role of E-cadherin.Reciprocal regulation of the cholinic phenotype and epithelial-mesenchymal transition in glioblastoma cells.ZEB1 Promotes Oxaliplatin Resistance through the Induction of Epithelial - Mesenchymal Transition in Colon Cancer Cells.Hypoxia-induced reactive oxygen species mediate N-cadherin and SERPINE1 expression, EGFR signalling and motility in MDA-MB-468 breast cancer cells.Transcription factor c-Myb inhibits breast cancer lung metastasis by suppression of tumor cell seeding.The crosstalk between p38 and Akt signaling pathways orchestrates EMT by regulating SATB2 expression in NSCLC cells.miR-190 suppresses breast cancer metastasis by regulation of TGF-β-induced epithelial-mesenchymal transition.
P2860
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P2860
Direct repression of MYB by ZEB1 suppresses proliferation and epithelial gene expression during epithelial-to-mesenchymal transition of breast cancer cells.
description
2013 nî lūn-bûn
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2013年の論文
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年学术文章
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2013年學術文章
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name
Direct repression of MYB by ZE ...... sition of breast cancer cells.
@en
Direct repression of MYB by ZE ...... sition of breast cancer cells.
@nl
type
label
Direct repression of MYB by ZE ...... sition of breast cancer cells.
@en
Direct repression of MYB by ZE ...... sition of breast cancer cells.
@nl
prefLabel
Direct repression of MYB by ZE ...... sition of breast cancer cells.
@en
Direct repression of MYB by ZE ...... sition of breast cancer cells.
@nl
P2093
P2860
P50
P356
P1476
Direct repression of MYB by ZE ...... sition of breast cancer cells.
@en
P2093
Bryce J W van Denderen
Cletus Pinto
Eliza T L Soo
Lloyd Pereira
N P A Devika Gunasinghe
Randy Suryadinata
Yvette Drabsch
P2860
P2888
P356
10.1186/BCR3580
P577
2013-11-27T00:00:00Z
P5875
P6179
1014796182