Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
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Tumor-Associated Macrophages and Neutrophils in Tumor MicroenvironmentComplement and macrophage crosstalk during process of angiogenesis in tumor progressionPattern-recognition receptors and gastric cancerTumor endothelial cellsThe interaction of anticancer therapies with tumor-associated macrophagesCirculating biomarkers in advanced renal cell carcinoma: clinical applicationsChanging perspective on oncometabolites: from metabolic signature of cancer to tumorigenic and immunosuppressive agentsOrchestration of angiogenesis by immune cellsAngiogenic capacity of M1- and M2-polarized macrophages is determined by the levels of TIMP-1 complexed with their secreted proMMP-9.Circulating giant macrophages as a potential biomarker of solid tumorsIntravital imaging reveals distinct responses of depleting dynamic tumor-associated macrophage and dendritic cell subpopulations.The NOD-like receptor signalling pathway in Helicobacter pylori infection and related gastric cancer: a case-control study and gene expression analyses.Human adipose tissue-resident monocytes exhibit an endothelial-like phenotype and display angiogenic propertiesSystemic Immune-Inflammation Index and Circulating T-Cell Immune Index Predict Outcomes in High-Risk Acral Melanoma Patients Treated with High-Dose InterferonRecruitment of myeloid but not endothelial precursor cells facilitates tumor regrowth after local irradiationBone marrow is a reservoir for proangiogenic myelomonocytic cells but not endothelial cells in spontaneous tumorsTissue-infiltrating neutrophils constitute the major in vivo source of angiogenesis-inducing MMP-9 in the tumor microenvironmentDistribution of bone-marrow-derived endothelial and immune cells in a murine colitis-associated colorectal cancer model.A think tank of TINK/TANKs: tumor-infiltrating/tumor-associated natural killer cells in tumor progression and angiogenesis.Direct evidence for lineage-dependent effects of bone marrow stromal cells on tumor progressionTumor-recruited neutrophils and neutrophil TIMP-free MMP-9 regulate coordinately the levels of tumor angiogenesis and efficiency of malignant cell intravasation.Angiogenic activity of breast cancer patients' monocytes reverted by combined use of systems modeling and experimental approachesAnti-VEGF treatment-resistant pancreatic cancers secrete proinflammatory factors that contribute to malignant progression by inducing an EMT cell phenotype.Myeloid angiogenic cells act as alternative M2 macrophages and modulate angiogenesis through interleukin-8.Activating transcription factor 4 promotes angiogenesis of breast cancer through enhanced macrophage recruitment.Tumour macrophages as potential targets of bisphosphonates.Roles of bone marrow cells in skeletal metastases: no longer bystandersIdentification of Reprogrammed Myeloid Cell Transcriptomes in NSCLCNG2 proteoglycan-dependent recruitment of tumor macrophages promotes pericyte-endothelial cell interactions required for brain tumor vascularization.Breast cancer cell uptake of the inflammatory mediator neutrophil elastase triggers an anticancer adaptive immune responseEarly vascular deficits are correlated with delayed mammary tumorigenesis in the MMTV-PyMT transgenic mouse following genetic ablation of the NG2 proteoglycan.Tumor-associated macrophages: function, phenotype, and link to prognosis in human lung cancer.TIE-2-expressing monocytes are lymphangiogenic and associate specifically with lymphatics of human breast cancerNotch1 regulates angio-supportive bone marrow-derived cells in mice: relevance to chemoresistance.Perivascular M2 Macrophages Stimulate Tumor Relapse after ChemotherapyObesity promotes breast cancer by CCL2-mediated macrophage recruitment and angiogenesis.Parathyroid hormone-related protein drives a CD11b+Gr1+ cell-mediated positive feedback loop to support prostate cancer growth.Tumor angiogenesis: MMP-mediated induction of intravasation- and metastasis-sustaining neovasculature.CD13-positive bone marrow-derived myeloid cells promote angiogenesis, tumor growth, and metastasis.NG2 proteoglycan promotes tumor vascularization via integrin-dependent effects on pericyte function.
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Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 18 February 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
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name
Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
@en
Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
@nl
type
label
Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
@en
Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
@nl
prefLabel
Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
@en
Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
@nl
P2093
P2860
P1476
Elusive identities and overlapping phenotypes of proangiogenic myeloid cells in tumors.
@en
P2093
Claire E Lewis
J Martin Brown
Michele De Palma
Napoleone Ferrara
Seth B Coffelt
P2860
P304
P356
10.2353/AJPATH.2010.090786
P407
P577
2010-02-18T00:00:00Z