GS domain mutations that constitutively activate T beta R-I, the downstream signaling component in the TGF-beta receptor complex.
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Direct interaction between Smad3, APC10, CDH1 and HEF1 in proteasomal degradation of HEF1Smad proteins function as co-modulators for MEF2 transcriptional regulatory proteinsThe adaptor molecule Disabled-2 links the transforming growth factor beta receptors to the Smad pathway.Mutations in bone morphogenetic protein receptor 1B cause brachydactyly type A2Transforming growth factor {beta} (TGF-{beta})-Smad target gene protein tyrosine phosphatase receptor type kappa is required for TGF-{beta} functionSmad3 allostery links TGF-beta receptor kinase activation to transcriptional controlActivation of signalling by the activin receptor complexING2 as a novel mediator of transforming growth factor-beta-dependent responses in epithelial cellsDual role of the Smad4/DPC4 tumor suppressor in TGFbeta-inducible transcriptional complexesSmad6 inhibits BMP/Smad1 signaling by specifically competing with the Smad4 tumor suppressorTranscriptional activating activity of Smad4: roles of SMAD hetero-oligomerization and enhancement by an associating transactivatorControl of prostate cell growth: BMP antagonizes androgen mitogenic activity with incorporation of MAPK signals in Smad1Serine and tyrosine phosphorylations cooperate in Raf-1, but not B-Raf activationTyrosine phosphorylation acts as a molecular switch to full-scale activation of the eIF2alpha RNA-dependent protein kinase.BF-1 interferes with transforming growth factor beta signaling by associating with Smad partnersSTRAP and Smad7 synergize in the inhibition of transforming growth factor beta signalingE2F and histone deacetylase mediate transforming growth factor beta repression of cdc25A during keratinocyte cell cycle arrestThe novel E3 ubiquitin ligase Tiul1 associates with TGIF to target Smad2 for degradationTLP, a novel modulator of TGF-beta signaling, has opposite effects on Smad2- and Smad3-dependent signalingSynergistic cooperation of TFE3 and smad proteins in TGF-beta-induced transcription of the plasminogen activator inhibitor-1 geneStructure of the bone morphogenetic protein receptor ALK2 and implications for fibrodysplasia ossificans progressivaACVR1B (ALK4, activin receptor type 1B) gene mutations in pancreatic carcinomaTGF-beta1 stimulates human AT1 receptor expression in lung fibroblasts by cross talk between the Smad, p38 MAPK, JNK, and PI3K signaling pathwaysHereditary haemorrhagic telangiectasia: current views on genetics and mechanisms of diseaseHuman type II receptor for bone morphogenic proteins (BMPs): extension of the two-kinase receptor model to the BMPsGADD34-PP1c recruited by Smad7 dephosphorylates TGFbeta type I receptorActivin receptor-like kinase 1 modulates transforming growth factor-beta 1 signaling in the regulation of angiogenesisThe Spemann organizer gene, Goosecoid, promotes tumor metastasisTransforming growth factor beta signaling impairs Neu-induced mammary tumorigenesis while promoting pulmonary metastasisGenome-wide microarray analysis of TGFbeta signaling in the Drosophila brainDifferential regulation of follicle stimulating hormone by activin A and TGFB1 in murine gonadotropes.The biological function of type I receptors of bone morphogenetic protein in boneClinical development of galunisertib (LY2157299 monohydrate), a small molecule inhibitor of transforming growth factor-beta signaling pathwayIntertwining of Activin A and TGFβ Signaling: Dual Roles in Cancer Progression and Cancer Cell InvasionAbnormal angiogenesis but intact hematopoietic potential in TGF-beta type I receptor-deficient miceThe NH2-terminal propeptide of type I procollagen acts intracellularly to modulate cell functionTranscriptome analyses of inhibitor-treated schistosome females provide evidence for cooperating Src-kinase and TGFβ receptor pathways controlling mitosis and eggshell formationCommunity structure detection for overlapping modules through mathematical programming in protein interaction networksNovel roles of Akt and mTOR in suppressing TGF-beta/ALK5-mediated Smad3 activationSmad8 mediates the signaling of the ALK-2 [corrected] receptor serine kinase
P2860
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P2860
GS domain mutations that constitutively activate T beta R-I, the downstream signaling component in the TGF-beta receptor complex.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on May 1995
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
GS domain mutations that const ...... the TGF-beta receptor complex.
@en
GS domain mutations that const ...... the TGF-beta receptor complex.
@nl
type
label
GS domain mutations that const ...... the TGF-beta receptor complex.
@en
GS domain mutations that const ...... the TGF-beta receptor complex.
@nl
prefLabel
GS domain mutations that const ...... the TGF-beta receptor complex.
@en
GS domain mutations that const ...... the TGF-beta receptor complex.
@nl
P2093
P2860
P1433
P1476
GS domain mutations that const ...... the TGF-beta receptor complex.
@en
P2093
P2860
P304
P407
P577
1995-05-01T00:00:00Z