Binge ethanol-drinking potentiates corticotropin releasing factor R1 receptor activity in the ventral tegmental area. - Wikidata - wikimedia - TriplyDB

Binge ethanol-drinking potentiates corticotropin releasing factor R1 receptor activity in the ventral tegmental area.

Binge ethanol-drinking potentiates corticotropin releasing factor R1 receptor activity in the ventral tegmental area.

http://www.wikidata.org/entity/Q37700982

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Diversity of Dopaminergic Neural Circuits in Response to Drug Exposure An Update on CRF Mechanisms Underlying Alcohol Use Disorders and Dependence Defining the role of corticotropin releasing factor binding protein in alcohol consumption.Alcohol and tobacco: how smoking may promote excessive drinking.NPY signaling inhibits extended amygdala CRF neurons to suppress binge alcohol drinking.Corticotropin Releasing Factor Binding Protein and CRF2 Receptors in the Ventral Tegmental Area: Modulation of Ethanol Binge Drinking in C57BL/6J Mice Rodent models and mechanisms of voluntary binge-like ethanol consumption: Examples, opportunities, and strategies for preclinical research.Preclinical evidence implicating corticotropin-releasing factor signaling in ethanol consumption and neuroadaptation.Nicotine administration in the cholinergic basal forebrain increases alcohol consumption in C57BL/6J mice.Extended Amygdala to Ventral Tegmental Area Corticotropin-Releasing Factor Circuit Controls Binge Ethanol Intake.Novel Roles for CRF-Binding Protein and CRF Receptor 2 in Binge Drinking.Binge Drinking Decreases Corticotropin-Releasing Factor-Binding Protein Expression in the Medial Prefrontal Cortex of Mice.Social stress-escalated intermittent alcohol drinking: modulation by CRF-R1 in the ventral tegmental area and accumbal dopamine in mice.Corticotropin-Releasing Factor (CRF) and Addictive Behaviors.The Corticotropin Releasing Factor Receptor 1 in Alcohol Use Disorder: Still a Valid Drug Target?

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Binge ethanol-drinking potentiates corticotropin releasing factor R1 receptor activity in the ventral tegmental area.

http://www.wikidata.org/entity/Q37700982

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 13 June 2013

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Binge ethanol-drinking potenti ...... in the ventral tegmental area.

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Binge ethanol-drinking potenti ...... in the ventral tegmental area.

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type

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Binge ethanol-drinking potenti ...... in the ventral tegmental area.

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Binge ethanol-drinking potenti ...... in the ventral tegmental area.

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Binge ethanol-drinking potenti ...... in the ventral tegmental area.

@en

Binge ethanol-drinking potenti ...... in the ventral tegmental area.

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Alcoholism: Clinical and Experimental Research

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Binge ethanol-drinking potenti ...... in the ventral tegmental area.

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Antonello Bonci

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Dennis R Sparta

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Dorit Ron

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Frederic Woodward Hopf

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Robert O Messing

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Saemi L Cho

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Stuart L Gibb

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P2860

Alcoholism: A Systems Approach From Molecular Physiology to Addictive Behavior

Corticotropin-releasing factor requires CRF binding protein to potentiate NMDA receptors via CRF receptor 2 in dopamine neurons

Two types of neurone in the rat ventral tegmental area and their synaptic inputs

Urocortins: CRF's siblings and their potential role in anxiety, depression and alcohol drinking behavior.

Magnitude of alcohol-related mortality and morbidity among U.S. college students ages 18-24: changes from 1998 to 2001

Corticotropin-releasing factor acting on corticotropin-releasing factor receptor type 1 is critical for binge alcohol drinking in mice.

"Binge" drinking experience in adolescent mice shows sex differences and elevated ethanol intake in adulthood.

Urocortin 1 microinjection into the mouse lateral septum regulates the acquisition and expression of alcohol consumption

Pathways to relapse: the neurobiology of drug- and stress-induced relapse to drug-taking.

Excitatory transmission from the amygdala to nucleus accumbens facilitates reward seeking.

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1680-1687

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scholarly article

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10.1111/ACER.12153

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10

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Garret D Stuber

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2013-06-13T00:00:00Z

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