Extracellular signal-regulated kinase (ERK) activation is required for itch sensation in the spinal cord.
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Neurochemical characterization of pERK-expressing spinal neurons in histamine-induced itch.Spinal neurons that contain gastrin-releasing peptide seldom express Fos or phosphorylate extracellular signal-regulated kinases in response to intradermal chloroquine.Role of ERK1/2 activation on itch sensation induced by bradykinin B1 activation in inflamed skin.Preprotachykinin A is expressed by a distinct population of excitatory neurons in the mouse superficial spinal dorsal horn including cells that respond to noxious and pruritic stimuli.Antioxidants Attenuate Acute and Chronic Itch: Peripheral and Central Mechanisms of Oxidative Stress in Pruritus.Spinal SET7/9 may contribute to the maintenance of cancer-induced bone pain in mice.The behavioral study on the interactive aggravation between pruritus and depression.NMDA receptor antagonists attenuate intrathecal morphine-induced pruritus through ERK phosphorylation.
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P2860
Extracellular signal-regulated kinase (ERK) activation is required for itch sensation in the spinal cord.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 03 April 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Extracellular signal-regulated ...... sensation in the spinal cord.
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type
label
Extracellular signal-regulated ...... sensation in the spinal cord.
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prefLabel
Extracellular signal-regulated ...... sensation in the spinal cord.
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P2093
P2860
P356
P1433
P1476
Extracellular signal-regulated ...... sensation in the spinal cord.
@en
P2093
Guan-Yu Jiang
Jia-Yin Chen
Ling Zhang
Ning-Jing Song
Qing-Xiu Wang
Ying Huang
Yu-Qiang Ding
P2860
P2888
P356
10.1186/1756-6606-7-25
P577
2014-04-03T00:00:00Z
P5875
P6179
1032066680