In vivo silencing of the transcription factor IRF5 reprograms the macrophage phenotype and improves infarct healing. - Wikidata - wikimedia - TriplyDB

In vivo silencing of the transcription factor IRF5 reprograms the macrophage phenotype and improves infarct healing.

In vivo silencing of the transcription factor IRF5 reprograms the macrophage phenotype and improves infarct healing.

http://www.wikidata.org/entity/Q37710952

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Inflammation and Inflammatory Cells in Myocardial Infarction and Reperfusion Injury: A Double-Edged Sword Immune Cells and Inflammation in Diabetic Nephropathy Modulators of Macrophage Polarization Influence Healing of the Infarcted Myocardium Monocytes in myocardial infarction Systemic and Cardiac Depletion of M2 Macrophage through CSF-1R Signaling Inhibition Alters Cardiac Function Post Myocardial Infarction Exosomes for Intramyocardial Intercellular Communication Lp-PLA2 Antagonizes Left Ventricular Healing After Myocardial Infarction by Impairing the Appearance of Reparative Macrophages Translational Nano-Medicines: Targeted Therapeutic Delivery for Cancer and Inflammatory Diseases It takes two to tango: monocyte and macrophage duality in the infarcted heart Sema3A promotes the resolution of cardiac inflammation after myocardial infarction An AAAG-Rich Oligodeoxynucleotide Rescues Mice from Bacterial Septic Peritonitis by Interfering Interferon Regulatory Factor 5.Regulatory T cells are recruited in the infarcted mouse myocardium and may modulate fibroblast phenotype and function.Regulation and consequences of monocytosis Cardiac macrophages and their role in ischaemic heart disease.IRF5 deficiency ameliorates lupus but promotes atherosclerosis and metabolic dysfunction in a mouse model of lupus-associated atherosclerosis Silencing collapsin response mediator protein-2 reprograms macrophage phenotype and improves infarct healing in experimental myocardial infarction model Sexually dimorphic outcomes and inflammatory responses in hypoxic-ischemic encephalopathy.Non-viral nanoparticle delivers small interfering RNA to macrophages in vitro and in vivo.Macrophages retain hematopoietic stem cells in the spleen via VCAM-1 Microglial and macrophage polarization—new prospects for brain repair Myocardial Infarction Activates CCR2(+) Hematopoietic Stem and Progenitor Cells.siRNA-based spherical nucleic acids reverse impaired wound healing in diabetic mice by ganglioside GM3 synthase knockdown.Endothelial cells suppress monocyte activation through secretion of extracellular vesicles containing antiinflammatory microRNAs.Inhibiting macrophage proliferation suppresses atherosclerotic plaque inflammation Beyond vascular inflammation--recent advances in understanding atherosclerosis.Arterial Hypertension Aggravates Innate Immune Responses after Experimental Stroke.Recent Advances in Targeted, Self-Assembling Nanoparticles to Address Vascular Damage Due to Atherosclerosis.Leukocytes Link Local and Systemic Inflammation in Ischemic Cardiovascular Disease: An Expanded "Cardiovascular Continuum".Interleukin-2/Anti-Interleukin-2 Immune Complex Attenuates Cardiac Remodeling after Myocardial Infarction through Expansion of Regulatory T Cells.Monocyte and macrophage contributions to cardiac remodeling.Specific detection of interferon regulatory factor 5 (IRF5): A case of antibody inequality.Macrophage Notch Ligand Delta-Like 4 Promotes Vein Graft Lesion Development: Implications for the Treatment of Vein Graft Failure.IRF5 governs liver macrophage activation that promotes hepatic fibrosis in mice and humans.PLGA-PNIPAM Microspheres Loaded with the Gastrointestinal Nutrient NaB Ameliorate Cardiac Dysfunction by Activating Sirt3 in Acute Myocardial Infarction Naoxintong attenuates Ischaemia/reperfusion Injury through inhibiting NLRP3 inflammasome activation."Pumping iron"-how macrophages handle iron at the systemic, microenvironmental, and cellular levels Targeted delivery systems for biological therapies of inflammatory diseases.Cellular immunity and cardiac remodeling after myocardial infarction: role of neutrophils, monocytes, and macrophages.Immune cell and other noncardiomyocyte regulation of cardiac hypertrophy and remodeling.Inflammation in cardiac injury, repair and regeneration.

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In vivo silencing of the transcription factor IRF5 reprograms the macrophage phenotype and improves infarct healing.

http://www.wikidata.org/entity/Q37710952

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 18 December 2013

@en

vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

In vivo silencing of the trans ...... and improves infarct healing.

@en

In vivo silencing of the trans ...... and improves infarct healing.

@nl

type

label

In vivo silencing of the trans ...... and improves infarct healing.

@en

In vivo silencing of the trans ...... and improves infarct healing.

@nl

prefLabel

In vivo silencing of the trans ...... and improves infarct healing.

@en

In vivo silencing of the trans ...... and improves infarct healing.

@nl

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J.JACC.2013.11.023

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Journal of the American College of Cardiology

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In vivo silencing of the trans ...... e and improves infarct healing

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Anna Borodovsky

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Benoit Tricot

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Boris Klebanov

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Daniel G Anderson

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Derrick Jeon

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Hendrik B Sager

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Jessica Truelove

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Kevin Fitzgerald

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Matthew Sebas

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Matthias Nahrendorf

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P2860

A common haplotype of interferon regulatory factor 5 (IRF5) regulates splicing and expression and is associated with increased risk of systemic lupus erythematosus

Progress toward in vivo use of siRNAs-II

Systemic leukocyte-directed siRNA delivery revealing cyclin D1 as an anti-inflammatory target

IRF5 promotes inflammatory macrophage polarization and TH1-TH17 responses

Integral role of IRF-5 in the gene induction programme activated by Toll-like receptors

Inflammation in atherosclerosis

Monocyte and macrophage heterogeneity

The healing myocardium sequentially mobilizes two monocyte subsets with divergent and complementary functions

Development of monocytes, macrophages, and dendritic cells

Macrophages in the pathogenesis of atherosclerosis

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1556-1566

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scholarly article

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10.1016/J.JACC.2013.11.023

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15

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63

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Gregory Wojtkiewicz

Gabriel Courties

Filip K Swirski

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2013-12-18T00:00:00Z

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259448155

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24361318

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