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Multifunctional Abl kinases in health and diseaseMechanisms of activation of the transcription factor Nrf2 by redox stressors, nutrient cues, and energy status and the pathways through which it attenuates degenerative diseaseNuclear Translocation of Crk Adaptor Proteins by the Influenza A Virus NS1 Protein.Defying c-Abl signaling circuits through small allosteric compounds.Attacking the supply wagons to starve cancer cells to death.Structure of the ABL2/ARG kinase in complex with dasatinibThe kinase ABL phosphorylates the microprocessor subunit DGCR8 to stimulate primary microRNA processing in response to DNA damage.Opium induces apoptosis in Jurkat cells via promotion of pro-apoptotic and inhibition of anti-apoptotic molecules.Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases.Phosphotyrosine profiling of curcumin-induced signaling.Nilotinib Effects in Parkinson's disease and Dementia with Lewy bodies.Coordination of signalling networks and tumorigenic properties by ABL in glioblastoma cells.The kinome 'at large' in cancer.TIE2-mediated tyrosine phosphorylation of H4 regulates DNA damage response by recruiting ABL1.Long-Term Side Effects of Tyrosine Kinase Inhibitors in Chronic Myeloid Leukemia.Ran Binding Protein 9 (RanBP9) is a novel mediator of cellular DNA damage response in lung cancer cells.Targeting ABL-IRE1α Signaling Spares ER-Stressed Pancreatic β Cells to Reverse Autoimmune Diabetes.Neutrophil FcγRIIA promotes IgG-mediated glomerular neutrophil capture via Abl/Src kinases.Mutant p53 dictates the oncogenic activity of c-Abl in triple-negative breast cancers.EnABLing microprocessor for apoptosis.VEGF165-induced vascular permeability requires NRP1 for ABL-mediated SRC family kinase activation.Src family kinase expression and subcellular localization in macrophages: implications for their role in CSF-1-induced macrophage migration.Long disordered regions of the C-terminal domain of Abelson tyrosine kinase have specific and additive functions in regulation and axon localization.c-Abl and Parkinson's Disease: Mechanisms and Therapeutic Potential.G-Protein Gα13 Functions with Abl Kinase to Regulate Actin Cytoskeletal Reorganization.Active site-adjacent phosphorylation at Tyr-397 by c-Abl kinase inactivates caspase-9.Reduction of Blood Amyloid-β Oligomers in Alzheimer's Disease Transgenic Mice by c-Abl Kinase Inhibition.Advances in studies of tyrosine kinase inhibitors and their acquired resistance.Receptor tyrosine kinases and downstream pathways as druggable targets for cancer treatment: the current arsenal of inhibitors.Chronic myeloid leukemia: the paradigm of targeting oncogenic tyrosine kinase signaling and counteracting resistance for successful cancer therapy.Optogenetically controlled protein kinases for regulation of cellular signaling.Amplification of F-Actin Disassembly and Cellular Repulsion by Growth Factor Signaling.Common effects of attractive and repulsive signaling: Further analysis of Mical-mediated F-actin disassembly and regulation by Abl.
P2860
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P2860
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 13 January 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The capable ABL: what is its biological function?
@ast
The capable ABL: what is its biological function?
@en
The capable ABL: what is its biological function?
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type
label
The capable ABL: what is its biological function?
@ast
The capable ABL: what is its biological function?
@en
The capable ABL: what is its biological function?
@nl
prefLabel
The capable ABL: what is its biological function?
@ast
The capable ABL: what is its biological function?
@en
The capable ABL: what is its biological function?
@nl
P2860
P356
P1476
The capable ABL: what is its biological function?
@en
P2093
Jean Y J Wang
P2860
P304
P356
10.1128/MCB.01454-13
P407
P577
2014-01-13T00:00:00Z