Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
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MicroRNA miR-214 Inhibits Snakehead Vesiculovirus Replication by Promoting IFN-α Expression via Targeting Host Adenosine 5'-Monophosphate-Activated Protein Kinase.Internal genes of a highly pathogenic H5N1 influenza virus determine high viral replication in myeloid cells and severe outcome of infection in mice.MicroRNA miR-214 Inhibits Snakehead Vesiculovirus Replication by Promoting IFN-α Expression via Targeting Host Adenosine 5′-Monophosphate-Activated Protein Kinase.Integrated Lung and Tracheal mRNA-Seq and miRNA-Seq Analysis of Dogs with an Avian-Like H5N1 Canine Influenza Virus Infection.Endogenous Cellular MicroRNAs Mediate Antiviral Defense against Influenza A Virus.Surviving Deadly Lung Infections: Innate Host Tolerance Mechanisms in the Pulmonary System.The Drivers of Pathology in Zoonotic Avian Influenza: The Interplay Between Host and Pathogen
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P2860
Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 10 March 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
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Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
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type
label
Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
@en
Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
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prefLabel
Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
@en
Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
@nl
P2093
P2860
P50
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Endothelial cell tropism is a determinant of H5N1 pathogenesis in mammalian species.
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Jasmine T Perez
John Steel
Randy A Albrecht
Smanla Tundup
Tamas Nagy
P2860
P304
P356
10.1371/JOURNAL.PPAT.1006270
P577
2017-03-10T00:00:00Z