Trichostatin A, a histone deacetylase inhibitor, suppresses proliferation and epithelial-mesenchymal transition in retinal pigment epithelium cells.
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Effect of histone deacetylase inhibitors trichostatin A and valproic acid on hair cell regeneration in zebrafish lateral line neuromastsThe complex interplay between ERK1/2, TGFβ/Smad, and Jagged/Notch signaling pathways in the regulation of epithelial-mesenchymal transition in retinal pigment epithelium cells.Cell models to study regulation of cell transformation in pathologies of retinal pigment epitheliumAttenuation of choroidal neovascularization by histone deacetylase inhibitor.Quantitative analysis of injury-induced anterior subcapsular cataract in the mouse: a model of lens epithelial cells proliferation and epithelial-mesenchymal transitionHistone Deacetylases Inhibitors in the Treatment of Retinal Degenerative Diseases: Overview and PerspectivesThe histone deacetylase inhibitor trichostatin a decreases lymphangiogenesis by inducing apoptosis and cell cycle arrest via p21-dependent pathwaysαB-crystallin is essential for the TGF-β2-mediated epithelial to mesenchymal transition of lens epithelial cellsQuercetin Inhibits Pulmonary Arterial Endothelial Cell Transdifferentiation Possibly by Akt and Erk1/2 Pathways.The roles of signaling pathways in epithelial-to-mesenchymal transition of PVR.HDACs and HDAC Inhibitors in Cancer Development and Therapy.Trichostatin A Inhibits Retinal Pigmented Epithelium Activation in an In Vitro Model of Proliferative Vitreoretinopathy.HDAC is essential for epigenetic regulation of Thy-1 gene expression during LPS/TLR4-mediated proliferation of lung fibroblasts.Suppressive effect of AMP-activated protein kinase on the epithelial-mesenchymal transition in retinal pigment epithelial cells.Trichostatin A inhibits radiation-induced epithelial-to-mesenchymal transition in the alveolar epithelial cells.Trichostatin A ameliorates renal tubulointerstitial fibrosis through modulation of the JNK-dependent Notch-2 signaling pathway.Myofibroblast transdifferentiation: The dark force in ocular wound healing and fibrosis.
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Trichostatin A, a histone deacetylase inhibitor, suppresses proliferation and epithelial-mesenchymal transition in retinal pigment epithelium cells.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 23 January 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Trichostatin A, a histone deac ...... inal pigment epithelium cells.
@en
Trichostatin A, a histone deac ...... inal pigment epithelium cells.
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type
label
Trichostatin A, a histone deac ...... inal pigment epithelium cells.
@en
Trichostatin A, a histone deac ...... inal pigment epithelium cells.
@nl
prefLabel
Trichostatin A, a histone deac ...... inal pigment epithelium cells.
@en
Trichostatin A, a histone deac ...... inal pigment epithelium cells.
@nl
P2093
P2860
P356
P1476
Trichostatin A, a histone deac ...... inal pigment epithelium cells.
@en
P2093
P2860
P304
P356
10.1111/JCMM.12212
P577
2014-01-23T00:00:00Z