about
Phyla- and Subtype-Selectivity of CgNa, a Na Channel Toxin from the Venom of the Giant Caribbean Sea Anemone Condylactis GiganteaThe search for novel analgesics: targets and mechanismsA SCN9A gene-encoded dorsal root ganglia sodium channel polymorphism associated with severe fibromyalgiaPrimary erythromelalgia: a reviewTheoretical and simulation studies on voltage-gated sodium channelsAMPK: An emerging target for modification of injury-induced pain plasticity.Mammalian Neuronal Sodium Channel Blocker μ-Conotoxin BuIIIB Has a Structured N-Terminus That Influences PotencyRoles of Voltage-Gated Tetrodotoxin-Sensitive Sodium Channels NaV1.3 and NaV1.7 in Diabetes and Painful Diabetic NeuropathySelective blocking effects of 4,9-anhydrotetrodotoxin, purified from a crude mixture of tetrodotoxin analogues, on NaV1.6 channels and its chemical aspectsA de novo gain-of-function mutation in SCN11A causes loss of pain perceptionRNA-Seq Analysis of Human Trigeminal and Dorsal Root Ganglia with a Focus on ChemoreceptorsSelective spider toxins reveal a role for the Nav1.1 channel in mechanical painSubtype-Selective Small Molecule Inhibitors Reveal a Fundamental Role for Nav1.7 in Nociceptor Electrogenesis, Axonal Conduction and Presynaptic Release.Voltage-gated sodium channel in grasshopper mice defends against bark scorpion toxinThe Concise Guide to PHARMACOLOGY 2013/14: ion channelsSodium overload due to a persistent current that attenuates the arrhythmogenic potential of a novel LQT3 mutationCorrelation of Nav1.8 and Nav1.9 sodium channel expression with neuropathic pain in human subjects with lingual nerve neuromas.Enhanced excitability of primary sensory neurons and altered gene expression of neuronal ion channels in dorsal root ganglion in paclitaxel-induced peripheral neuropathyMutations at opposite ends of the DIII/S4-S5 linker of sodium channel Na V 1.7 produce distinct pain disordersIsolation and characterization of CvIV4: a pain inducing α-scorpion toxin.Synthesis and analgesic effects of μ-TRTX-Hhn1b on models of inflammatory and neuropathic pain.In vivo gene knockdown in rat dorsal root ganglia mediated by self-complementary adeno-associated virus serotype 5 following intrathecal delivery.Tetrodotoxin (TTX) as a therapeutic agent for pain.PKCε phosphorylation of the sodium channel NaV1.8 increases channel function and produces mechanical hyperalgesia in mice.Gain-of-function Nav1.8 mutations in painful neuropathy.mTORC1 inhibition induces pain via IRS-1-dependent feedback activation of ERK.Topical analgesics for neuropathic pain: preclinical exploration, clinical validation, future development.Sea-anemone toxin ATX-II elicits A-fiber-dependent pain and enhances resurgent and persistent sodium currents in large sensory neurons.VEGF induces sensory and motor peripheral plasticity, alters bladder function, and promotes visceral sensitivity.Control of somatic membrane potential in nociceptive neurons and its implications for peripheral nociceptive transmission.Functional upregulation of nav1.8 sodium channels on the membrane of dorsal root Ganglia neurons contributes to the development of cancer-induced bone painSubgroup-elimination transcriptomics identifies signaling proteins that define subclasses of TRPV1-positive neurons and a novel paracrine circuit.Role for monocyte chemoattractant protein-1 in the induction of chronic muscle pain in the ratNav1.7 is the predominant sodium channel in rodent olfactory sensory neurons.Comprehensive RNA-Seq expression analysis of sensory ganglia with a focus on ion channels and GPCRs in Trigeminal gangliaOxidation-sensitive nociception involved in endometriosis-associated pain.Expanding chemical diversity of conotoxins: peptoid-peptide chimeras of the sodium channel blocker μ-KIIIA and its selenopeptide analoguesFunctional properties and toxin pharmacology of a dorsal root ganglion sodium channel viewed through its voltage sensorsIncreased expression of sodium channel subunit Nav1.1 in the injured dorsal root ganglion after peripheral nerve injuryMechanism of μ-conotoxin PIIIA binding to the voltage-gated Na+ channel NaV1.4.
P2860
Q21129291-CD02EDA2-534B-4B2E-AF8C-C629745409E7Q24288817-EC93D931-18B5-40D5-9D19-49EB9571CDD6Q24611164-310A3C36-6609-4B3C-97F3-0364763F782DQ26784432-B726EC44-B93B-4A96-BABE-2DCD2B14B4E6Q26998280-6E16B937-4580-4F66-847F-34B6B80EF731Q27021903-008B52B0-D0A3-4C97-9FF4-C1E5F8E50172Q27677218-BF8103DF-2B4E-4EFF-AAC3-AAC431DAB2BEQ28072725-D25FD8F1-94FC-4BF2-9F2D-8BBF6ADFA90CQ28084324-6FB5AD03-D60F-4E9B-82C2-5C4E7BF6990FQ28298515-7B61DB32-E80E-427D-B28F-249798A6858AQ28548290-055A2307-2B65-4A14-AD44-F176ED0C0916Q28594552-1F515DF3-BF98-43D3-848C-A80D460993B2Q28601095-A76A9849-3632-4672-9611-CA2269C7EE7DQ28655574-462DAC5D-4FE6-4B44-8BAD-1CD13053E0F8Q30486735-73C5CA12-33D9-4116-BF70-A5E30AE9FF5CQ33163356-DBBE627F-0AEB-4C83-9146-855B8B35F32BQ33591220-7F56533D-6B95-4F64-8088-926F30310DD9Q33650951-E02BC8D5-6A30-4E72-9B18-56D1E2213532Q33875626-C17DD728-178E-463B-B625-C7ED93C4ACBCQ34009359-76BB4B82-C513-4BD7-9B3E-744C90CAB119Q34101792-5EB3138E-B69C-4673-9CB4-47B2D6A3F105Q34189539-80EF6EEA-7BAF-45CD-A9B8-075F4F7C0025Q34260913-BC1AEF2B-6A60-408D-831F-70F68961FE7FQ34262020-58F6EB5A-F7B8-46E8-A3A3-C8B45C4B49E6Q34309236-F92A1422-3E58-46AC-970A-0DBDC4595F4CQ34340709-DEC26013-2577-45A0-B0CB-733A48FEE709Q34376548-F9063532-8BFA-40B0-BDAB-9CD39866B126Q34414770-FCDAEA4F-C304-4DE6-8EDE-AEC5F2655832Q34515175-0F83C3AA-6100-4017-A651-FF7BD1134A36Q34585723-D0672921-03DF-4223-8341-E23A4D4DFA09Q34682051-261B8F4E-1FC1-4249-91B9-1857C2C12370Q34796577-D39DF8F6-B266-4756-A5D3-DC8BC3B9BA88Q35001326-033CD502-6B05-4016-98F6-3C61EA74DC1BQ35003780-2C663590-D33E-4054-AC5A-622A8BF8FBC9Q35048508-E32BBD7C-860B-4577-A7D9-7B50377CF901Q35085111-C5997246-65D5-4C54-A3E7-9BF8AF78C5F9Q35095160-3E971856-F2CF-43CC-A6F6-A3EA44E0F7E9Q35102163-1E6A95DA-8D26-49B5-8CD4-6D8FAE703C99Q35118785-DA013E9A-13BC-4E3F-AACC-063B83D52ADDQ35133771-10E71DDA-318C-43A1-97E0-FCDD107300F7
P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Sodium channels in normal and pathological pain.
@en
Sodium channels in normal and pathological pain.
@nl
type
label
Sodium channels in normal and pathological pain.
@en
Sodium channels in normal and pathological pain.
@nl
prefLabel
Sodium channels in normal and pathological pain.
@en
Sodium channels in normal and pathological pain.
@nl
P2093
P1476
Sodium channels in normal and pathological pain.
@en
P2093
Joel A Black
Stephen G Waxman
Sulayman D Dib-Hajj
Theodore R Cummins
P304
P356
10.1146/ANNUREV-NEURO-060909-153234
P577
2010-01-01T00:00:00Z