The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
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Gut Microbiota and Lifestyle Interventions in NAFLDNegative, Null and Beneficial Effects of Drinking Water on Energy Intake, Energy Expenditure, Fat Oxidation and Weight Change in Randomized Trials: A Qualitative ReviewChemistry and biochemistry of 13C hyperpolarized magnetic resonance using dynamic nuclear polarizationIs Western Diet-Induced Nonalcoholic Steatohepatitis in Ldlr-/- Mice Reversible?Molecular Pathogenesis of NASHHow Useful Are Monogenic Rodent Models for the Study of Human Non-Alcoholic Fatty Liver Disease?Bifunctional homodimeric triokinase/FMN cyclase: contribution of protein domains to the activities of the human enzyme and molecular dynamics simulation of domain movementsNon-alcoholic fatty liver disease: need for a balanced nutritional sourceRole of Uric Acid Metabolism-Related Inflammation in the Pathogenesis of Metabolic Syndrome Components Such as Atherosclerosis and Nonalcoholic SteatohepatitisWhat is metabolic syndrome, and why are children getting it?Reactive oxygen species-induced TXNIP drives fructose-mediated hepatic inflammation and lipid accumulation through NLRP3 inflammasome activationEndocrine-disrupting chemicals and fatty liver disease.The Sweet Path to Metabolic Demise: Fructose and Lipid SynthesisThe Riddle of Nonalcoholic Fatty Liver Disease: Progression From Nonalcoholic Fatty Liver to Nonalcoholic Steatohepatitis.Fructose: it's "alcohol without the buzz".Mitoneet mediates TNFα-induced necroptosis promoted by exposure to fructose and ethanol.Elevated serum uric acid levels are associated with non-alcoholic fatty liver disease independently of metabolic syndrome features in the United States: Liver ultrasound data from the National Health and Nutrition Examination Survey.The relationship of sugar to population-level diabetes prevalence: an econometric analysis of repeated cross-sectional dataProlonged exposure to low-dose microcystin induces nonalcoholic steatohepatitis in mice: a systems toxicology study.Lifetime Exposure to a Constant Environment Amplifies the Impact of a Fructose-Rich Diet on Glucose Homeostasis during PregnancyHigh Dietary Fructose: Direct or Indirect Dangerous Factors Disturbing Tissue and Organ FunctionspNaKtide Attenuates Steatohepatitis and Atherosclerosis by Blocking Na/K-ATPase/ROS Amplification in C57Bl6 and ApoE Knockout Mice Fed a Western Diet.Glucose transporter 8 (GLUT8) mediates fructose-induced de novo lipogenesis and macrosteatosis.Xyloketal B Attenuates Fatty Acid-Induced Lipid Accumulation via the SREBP-1c Pathway in NAFLD Models.Hepatic glucose sensing is impaired, but can be normalized, in people with impaired fasting glucose.Toxic AGE (TAGE) Theory for the Pathophysiology of the Onset/Progression of NAFLD and ALDExperimental models of non-alcoholic fatty liver disease in rats.The effects of fructose-containing sugars on weight, body composition and cardiometabolic risk factors when consumed at up to the 90th percentile population consumption level for fructoseDiffering endoplasmic reticulum stress response to excess lipogenesis versus lipid oversupply in relation to hepatic steatosis and insulin resistance.Emodin prevents intrahepatic fat accumulation, inflammation and redox status imbalance during diet-induced hepatosteatosis in rats.Approach to assessing determinants of glucose homeostasis in the conscious mouseToward a unifying hypothesis of metabolic syndrome.Comparison with ancestral diets suggests dense acellular carbohydrates promote an inflammatory microbiota, and may be the primary dietary cause of leptin resistance and obesityEnergy and fructose from beverages sweetened with sugar or high-fructose corn syrup pose a health risk for some peopleJNK regulation of hepatic manifestations of the metabolic syndrome.Fructose, pregnancy and later life impacts.Effect of fructose on markers of non-alcoholic fatty liver disease (NAFLD): a systematic review and meta-analysis of controlled feeding trials.Rodent models for metabolic syndrome research.Pnpla3/Adiponutrin deficiency in mice does not contribute to fatty liver disease or metabolic syndrome.Subclinical Diabetes.
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The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 06 April 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
@en
The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
@nl
type
label
The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
@en
The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
@nl
prefLabel
The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
@en
The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
@nl
P2093
P356
P1476
The role of fructose in the pathogenesis of NAFLD and the metabolic syndrome.
@en
P2093
Annie Valente
Jean-Marc Schwarz
Jung Sub Lim
Michele Mietus-Snyder
Robert H Lustig
P2888
P304
P356
10.1038/NRGASTRO.2010.41
P407
P577
2010-04-06T00:00:00Z
P5875
P6179
1004432312