The Spontaneous Autoimmune Neuromyopathy in ICOSL-/- NOD Mice Is CD4+ T-Cell and Interferon-γ Dependent

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The Spontaneous Autoimmune Neuromyopathy in ICOSL-/- NOD Mice Is CD4+ T-Cell and Interferon-γ Dependent

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scientific article published on 31 March 2017

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The Spontaneous Autoimmune Neu ...... ell and Interferon-γ Dependent

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The Spontaneous Autoimmune Neu ...... ll and Interferon-γ Dependent.

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The Spontaneous Autoimmune Neu ...... ell and Interferon-γ Dependent

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The Spontaneous Autoimmune Neu ...... ll and Interferon-γ Dependent.

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The Spontaneous Autoimmune Neu ...... ell and Interferon-γ Dependent

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The Spontaneous Autoimmune Neu ...... ll and Interferon-γ Dependent.

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The Spontaneous Autoimmune Neu ...... ell and Interferon-γ Dependent

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Chantal Becourt

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Charbel Massaad

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Christophe Arnoult

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Claire Briet

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Gwladys Bourdenet

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Isabelle Tardivel

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Jean-Claude do Rego

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Laurent Drouot

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Nicolas Prevot

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P2860

B7/CD28 costimulation is essential for the homeostasis of the CD4+CD25+ immunoregulatory T cells that control autoimmune diabetes

Autoimmune dilated cardiomyopathy in PD-1 receptor-deficient mice

The murine autoimmune diabetes model: NOD and related strains

Congenic mapping of the type 1 diabetes locus, Idd3, to a 780-kb region of mouse chromosome 3: identification of a candidate segment of ancestral DNA by haplotype mapping.

Prevention of diabetes in nonobese diabetic mice by anti-I-A monoclonal antibodies: transfer of protection by splenic T cells

Distinct genetic control of autoimmune neuropathy and diabetes in the non-obese diabetic background.

Complexities of CD28/B7: CTLA-4 costimulatory pathways in autoimmunity and transplantation.

Autoantibodies against cardiac troponin I are responsible for dilated cardiomyopathy in PD-1-deficient mice.

Genetic heterogeneity of autoimmune disorders in the nonobese diabetic mouse.

Thinking bedside at the bench: the NOD mouse model of T1DM.

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287

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scholarly article

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10.3389/FIMMU.2017.00287

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Christian Boitard

Olivier Boyer

Ute Christine Rogner

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2017-03-31T00:00:00Z

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28424681

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5371727

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