Pleural fluid from tuberculous pleurisy inhibits the functions of T cells and the differentiation of Th1 cells via immunosuppressive factors. - Wikidata - wikimedia - TriplyDB

Pleural fluid from tuberculous pleurisy inhibits the functions of T cells and the differentiation of Th1 cells via immunosuppressive factors.

Pleural fluid from tuberculous pleurisy inhibits the functions of T cells and the differentiation of Th1 cells via immunosuppressive factors.

http://www.wikidata.org/entity/Q37729535

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New Insights into IDO Biology in Bacterial and Viral Infections.Alteration of naïve and memory B-cell subset in chronic graft-versus-host disease patients after treatment with mesenchymal stromal cells.M. tuberculosis induces potent activation of IDO-1, but this is not essential for the immunological control of infection.Serum indoleamine 2,3-dioxygenase activity predicts prognosis of pulmonary tuberculosis Immunometabolism within the tuberculosis granuloma: amino acids, hypoxia, and cellular respiration.Addressing the tuberculosis-depression syndemic to end the tuberculosis epidemic.A diagnostic algorithm for tuberculous pleurisy using the ELISPOT assay on peripheral blood and pleural effusion.

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Pleural fluid from tuberculous pleurisy inhibits the functions of T cells and the differentiation of Th1 cells via immunosuppressive factors.

http://www.wikidata.org/entity/Q37729535

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 24 January 2011

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Pleural fluid from tuberculous ...... via immunosuppressive factors.

@en

Pleural fluid from tuberculous ...... via immunosuppressive factors.

@nl

type

label

Pleural fluid from tuberculous ...... via immunosuppressive factors.

@en

Pleural fluid from tuberculous ...... via immunosuppressive factors.

@nl

prefLabel

Pleural fluid from tuberculous ...... via immunosuppressive factors.

@en

Pleural fluid from tuberculous ...... via immunosuppressive factors.

@nl

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Cellular and Molecular Immunology

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Pleural fluid from tuberculous ...... via immunosuppressive factors.

@en

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Changyou Wu

http://www.w3.org/2001/XMLSchema#string

Dan Qiao

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Fengyu Huang

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Hui Wang

http://www.w3.org/2001/XMLSchema#string

Li Li

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Qin Li

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Suihua Lao

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Xiaoying Fu

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Yun Liu

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P2860

Role of A2a extracellular adenosine receptor-mediated signaling in adenosine-mediated inhibition of T-cell activation and expansion

IDO expression by dendritic cells: tolerance and tryptophan catabolism

Differentiation of effector CD4 T cell populations (*)

A2A adenosine receptor induction inhibits IFN-gamma production in murine CD4+ T cells.

Activation of A2A adenosine receptor attenuates intestinal inflammation in animal models of inflammatory bowel disease.

A2A receptor signaling promotes peripheral tolerance by inducing T-cell anergy and the generation of adaptive regulatory T cells.

Dynamic changes in pro- and anti-inflammatory cytokine profiles and gamma interferon receptor signaling integrity correlate with tuberculosis disease activity and response to curative treatment.

Cytokine production at the site of disease in human tuberculosis

Endogenous kynurenines as targets for drug discovery and development.

Pleural effusions: the diagnostic separation of transudates and exudates.

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172-180

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scholarly article

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10.1038/CMI.2010.80

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2

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8

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2011-01-24T00:00:00Z

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49777562

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21258363

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