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Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

http://www.wikidata.org/entity/Q37733614

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Intravenous pretreatment with emulsified isoflurane preconditioning protects kidneys against ischemia/reperfusion injury in rats.Clinical Risk Scoring Models for Prediction of Acute Kidney Injury after Living Donor Liver Transplantation: A Retrospective Observational Study.No Effect of Remote Ischemic Conditioning Strategies on Recovery from Renal Ischemia-Reperfusion Injury and Protective Molecular Mediators Heme Oxygenase-1 Regulates Myeloid Cell Trafficking in AKI Economic Evaluation of Pharmacologic Pre- and Postconditioning With Sevoflurane Compared With Total Intravenous Anesthesia in Liver Surgery: A Cost Analysis.Immune Modulation by Volatile Anesthetics Inhaled nitric oxide prevents NSAID-induced renal impairment in pseudo-normovolaemic piglets.Case report: proximal tubule impairment following volatile anesthetic exposure.A Review of Anesthetic Effects on Renal Function: Potential Organ Protection.Volatile sedation in the intensive care unit: A systematic review and meta-analysis.Renal complications of anaesthesia.Expression of Kidney Injury Molecule-1 in Healthy and Diseased Feline Kidney Tissue.

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Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

http://www.wikidata.org/entity/Q37733614

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 07 February 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

@en

Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

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type

label

Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

@en

Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

@nl

prefLabel

Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

@en

Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

@nl

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Journal of the American Society of Nephrology

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Volatile anesthetics and AKI: risks, mechanisms, and a potential therapeutic window.

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H Thomas Lee

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Kyota Fukazawa

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P2860

Methoxyflurane nephrotoxicity. A study of dose response in man

Nephrotoxicity associated with methoxyflurane anesthesia

Phosphatidylserine-dependent ingestion of apoptotic cells promotes TGF-beta1 secretion and the resolution of inflammation

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

Sphingosine-1-phosphate: an enigmatic signalling lipid

Regulatory T cells in AKI.

Autocrine adenosine signaling promotes regulatory T cell-mediated renal protection.

Regulatory T cells suppress innate immunity in kidney ischemia-reperfusion injury

Adenosine A2A receptor activation reduces hepatic ischemia reperfusion injury by inhibiting CD1d-dependent NKT cell activation.

Isoflurane preconditioning decreases myocardial infarction in rabbits via up-regulation of hypoxia inducible factor 1 that is mediated by mammalian target of rapamycin.

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884-892

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scholarly article

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10.1681/ASN.2013111215

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5

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25

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2014-02-07T00:00:00Z

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24511126

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4005317

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