Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
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Communication in the heart: the role of the innate immune system in coordinating cellular responses to ischemic injuryCan the TLR-4-mediated signaling pathway be "a key inflammatory promoter for sporadic TAA"?Understanding the expression of Toll-like receptors in Asian Indians predisposed to coronary artery disease.Are the leukocyte telomere length attrition and telomerase activity alteration potential predictor biomarkers for sporadic TAA in aged individuals?Activation of T lymphocytes in atherosclerotic plaques.Mechanisms of triglyceride accumulation in activated macrophagesIdiopathic inflammatory myopathies: pathogenic mechanisms of muscle weaknessDevelopmental onset of bilirubin-induced neurotoxicity involves Toll-like receptor 2-dependent signaling in humanized UDP-glucuronosyltransferase1 mice.Vascular smooth muscle cell motility: From migration to invasion.Innate immune signaling in cardiac ischemia.The emerging role of innate immunity in the heart and vascular system: for whom the cell tolls.Intrinsic danger: activation of Toll-like receptors in rheumatoid arthritis.Future potential biomarkers for postinterventional restenosis and accelerated atherosclerosis.Positioning of inflammatory biomarkers in the heart failure landscape.Innate immune receptors in heart failure: Side effect or potential therapeutic target?Smart mechanosensing machineries enable migration of vascular smooth muscle cells in atherosclerosis-relevant 3D matrices.Toll-like receptor 4 mediates vascular remodeling in hyperhomocysteinemia.Fibronectin III 13-14 domains induce joint damage via Toll-like receptor 4 activation and synergize with interleukin-1 and tumour necrosis factor.Toll-like receptor 4 mutation suppresses hyperhomocysteinemia-induced hypertension.Platelet Ca(2+) responses coupled to glycoprotein VI and Toll-like receptors persist in the presence of endothelial-derived inhibitors: roles for secondary activation of P2X1 receptors and release from intracellular Ca(2+) storesAnti-Inflamm-Ageing and/or Anti-Age-Related Disease Emerging Treatments: A Historical Alchemy or Revolutionary Effective Procedures?Toll-Like Receptors, Inflammation, and Calcific Aortic Valve Disease.
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Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 30 April 2010
@en
vedecký článok
@sk
vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
@en
Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
@nl
type
label
Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
@en
Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
@nl
prefLabel
Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
@en
Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
@nl
P2093
P921
P356
P1476
Endogenous inflammatory molecules engage Toll-like receptors in cardiovascular disease.
@en
P2093
Dominique P V de Kleijn
Fatih Arslan
Mihaela G Ionita
P304
P356
10.1159/000314270
P577
2010-04-30T00:00:00Z