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GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

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Role of SREBPs in Liver Diseases: A Mini-review

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GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

http://www.wikidata.org/entity/Q37747068

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 11 April 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

@da

vědecký článek

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name

GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

@en

GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

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type

label

GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

@en

GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

@nl

prefLabel

GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

@en

GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation.

@nl

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FMICB.2017.00576

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Frontiers in Microbiology

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GRIM-19 Restricts HCV Replication by Attenuating Intracellular Lipid Accumulation

@en

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Dong J Park

http://www.w3.org/2001/XMLSchema#string

Eun B Lee

http://www.w3.org/2001/XMLSchema#string

Jung-Hee Kim

http://www.w3.org/2001/XMLSchema#string

Marc P Windisch

http://www.w3.org/2001/XMLSchema#string

Pil S Sung

http://www.w3.org/2001/XMLSchema#string

Seung K Yoon

http://www.w3.org/2001/XMLSchema#string

Wonhee Hur

http://www.w3.org/2001/XMLSchema#string

P2860

Identification of GRIM-19, a novel cell death-regulatory gene induced by the interferon-beta and retinoic acid combination, using a genetic approach

Expression of hepatitis C virus proteins induces distinct membrane alterations including a candidate viral replication complex

Viral interferon regulatory factor 1 of Kaposi's sarcoma-associated herpesvirus interacts with a cell death regulator, GRIM19, and inhibits interferon/retinoic acid-induced cell death.

Hepatitis C virus hijacks host lipid metabolism

Nonlinear partial differential equations and applications: Genomic analysis of the host response to hepatitis C virus infection

Hepatitis C Virus Induces Proteolytic Cleavage of Sterol Regulatory Element Binding Proteins and Stimulates Their Phosphorylation via Oxidative Stress

Activation of STAT3 by the Hepatitis C Virus Core Protein Leads to Cellular Transformation

Fatty acid synthase is up-regulated during hepatitis C virus infection and regulates hepatitis C virus entry and production

Hepatitis C Virus Nonstructural 4B Protein Modulates Sterol Regulatory Element-binding Protein Signaling via the AKT Pathway

A genome-wide genetic screen for host factors required for hepatitis C virus propagation

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576

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scholarly article

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10.3389/FMICB.2017.00576

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8

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2017-04-11T00:00:00Z

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5387058

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