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Preclinical studies for gene therapy of Duchenne muscular dystrophy.

Preclinical studies for gene therapy of Duchenne muscular dystrophy.

http://www.wikidata.org/entity/Q37760380

about

Novel mutation in spectrin-like repeat 1 of dystrophin central domain causes protein misfolding and mild Becker muscular dystrophy.Activation of AKT signaling promotes cell growth and survival in α7β1 integrin-mediated alleviation of muscular dystrophy.

P2860

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Preclinical studies for gene therapy of Duchenne muscular dystrophy.

http://www.wikidata.org/entity/Q37760380

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 24 May 2010

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Preclinical studies for gene therapy of Duchenne muscular dystrophy.

@en

Preclinical studies for gene therapy of Duchenne muscular dystrophy.

@nl

type

label

Preclinical studies for gene therapy of Duchenne muscular dystrophy.

@en

Preclinical studies for gene therapy of Duchenne muscular dystrophy.

@nl

prefLabel

Preclinical studies for gene therapy of Duchenne muscular dystrophy.

@en

Preclinical studies for gene therapy of Duchenne muscular dystrophy.

@nl

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0883073810371006

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Journal of Child Neurology

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Preclinical studies for gene therapy of Duchenne muscular dystrophy

@en

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Glen B Banks

http://www.w3.org/2001/XMLSchema#string

Guy L Odom

http://www.w3.org/2001/XMLSchema#string

Jeffrey S Chamberlain

http://www.w3.org/2001/XMLSchema#string

P2860

Gene targeting in stem cells from individuals with osteogenesis imperfecta

Adeno-associated virus type 6 (AAV6) vectors mediate efficient transduction of airway epithelial cells in mouse lungs compared to that of AAV2 vectors

Novel adeno-associated viruses from rhesus monkeys as vectors for human gene therapy

ADENOVIRUS-ASSOCIATED DEFECTIVE VIRUS PARTICLES

Dystrophin: the protein product of the Duchenne muscular dystrophy locus

Complete cloning of the duchenne muscular dystrophy (DMD) cDNA and preliminary genomic organization of the DMD gene in normal and affected individuals

Microutrophin delivery through rAAV6 increases lifespan and improves muscle function in dystrophic dystrophin/utrophin-deficient mice

Prevention of dystrophic pathology in severely affected dystrophin/utrophin-deficient mice by morpholino-oligomer-mediated exon-skipping.

rAAV6-microdystrophin preserves muscle function and extends lifespan in severely dystrophic mice.

Truncated dystrophins can influence neuromuscular synapse structure.

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1149-1157

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scholarly article

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10.1177/0883073810371006

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9

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25

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2010-05-24T00:00:00Z

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