Electrophysiology and beyond: multiple roles of Na+ channel β subunits in development and disease.
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Trafficking mechanisms underlying neuronal voltage-gated ion channel localization at the axon initial segmentIon channels as drug targets in central nervous system disordersNa+ channel function, regulation, structure, trafficking and sequestrationNode of Ranvier disruption as a cause of neurological diseasesCrystal Structure and Molecular Imaging of the Nav Channel 3 Subunit Indicates a Trimeric AssemblyMechanisms of Action of Antiseizure Drugs and the Ketogenic DietSplice variants of Na(V)1.7 sodium channels have distinct β subunit-dependent biophysical propertiesPolymer-free optode nanosensors for dynamic, reversible, and ratiometric sodium imaging in the physiological rangeThe sorting receptor Rer1 controls Purkinje cell function via voltage gated sodium channels.Characterization of the axon initial segment (AIS) of motor neurons and identification of a para-AIS and a juxtapara-AIS, organized by protein 4.1B.Role of the axonal initial segment in psychiatric disorders: function, dysfunction, and interventionNeurotoxins and their binding areas on voltage-gated sodium channels.Sodium channel SCN8A (Nav1.6): properties and de novo mutations in epileptic encephalopathy and intellectual disability.The β1-subunit of Na(v)1.5 cardiac sodium channel is required for a dominant negative effect through α-α interaction.Atrial Fibrillation and SCN5A Variants.Independent and joint modulation of rat Nav1.6 voltage-gated sodium channels by coexpression with the auxiliary β1 and β2 subunitsNa+ channel Scn1b gene regulates dorsal root ganglion nociceptor excitability in vivoIdentification of Navβ1 residues involved in the modulation of the sodium channel Nav1.4.N-methyl-D-aspartate receptors mediate activity-dependent down-regulation of potassium channel genes during the expression of homeostatic intrinsic plasticity.Navβ subunits modulate the inhibition of Nav1.8 by the analgesic gating modifier μO-conotoxin MrVIBGene splicing of an invertebrate beta subunit (LCavβ) in the N-terminal and HOOK domains and its regulation of LCav1 and LCav2 calcium channels.Actions of Tefluthrin on Rat Na(v)1.7 Voltage-Gated Sodium Channels Expressed in Xenopus Oocytes.Na Channel β Subunits: Overachievers of the Ion Channel Family.Alternative splicing modulates inactivation of type 1 voltage-gated sodium channels by toggling an amino acid in the first S3-S4 linker.Subcellular heterogeneity of sodium current properties in adult cardiac ventricular myocytesVoltage-gated Na+ channel β1B: a secreted cell adhesion molecule involved in human epilepsyTranscriptomic Analysis of Induced Pluripotent Stem Cells Derived from Patients with Bipolar Disorder from an Old Order Amish Pedigree.The in vitro mechanisms and in vivo efficacy of intravenous lidocaine on the neuroinflammatory response in acute and chronic pain.Coexpression with Auxiliary β Subunits Modulates the Action of Tefluthrin on Rat Na(v)1.6 and Na(v)1.3 Sodium ChannelsThe sodium channel accessory subunit Navβ1 regulates neuronal excitability through modulation of repolarizing voltage-gated K⁺ channels.Biophysical and Pharmacological Characterization of Nav1.9 Voltage Dependent Sodium Channels Stably Expressed in HEK-293 Cells.Inhibition of voltage-gated Na(+) currents in sensory neurones by the sea anemone toxin APETx2.Voltage-gated sodium channels and metastatic disease.Effects of the β1 auxiliary subunit on modification of Rat Na(v)1.6 sodium channels expressed in HEK293 cells by the pyrethroid insecticides tefluthrin and deltamethrin.Electrophysiological characterization of Grueneberg ganglion olfactory neurons: spontaneous firing, sodium conductance, and hyperpolarization-activated currents.Abnormal neuronal patterning occurs during early postnatal brain development of Scn1b-null mice and precedes hyperexcitability.Transcriptional expression of voltage-gated Na⁺ and voltage-independent K⁺ channels in the developing rat superficial dorsal horn.Co-expression of Na(V)β subunits alters the kinetics of inhibition of voltage-gated sodium channels by pore-blocking μ-conotoxins.Sodium-calcium exchangers in rat trigeminal ganglion neurons.β1-C121W Is Down But Not Out: Epilepsy-Associated Scn1b-C121W Results in a Deleterious Gain-of-Function.
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Electrophysiology and beyond: multiple roles of Na+ channel β subunits in development and disease.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 23 June 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Electrophysiology and beyond: ...... ts in development and disease.
@en
Electrophysiology and beyond: ...... ts in development and disease.
@nl
type
label
Electrophysiology and beyond: ...... ts in development and disease.
@en
Electrophysiology and beyond: ...... ts in development and disease.
@nl
prefLabel
Electrophysiology and beyond: ...... ts in development and disease.
@en
Electrophysiology and beyond: ...... ts in development and disease.
@nl
P2860
P1433
P1476
Electrophysiology and beyond: ...... ts in development and disease.
@en
P2093
Gustavo A Patino
P2860
P356
10.1016/J.NEULET.2010.06.050
P407
P50
P577
2010-06-23T00:00:00Z