Destructive and protective roles of cytokines in periodontitis: a re-appraisal from host defense and tissue destruction viewpoints.
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Periodontal disease: linking the primary inflammation to bone lossHost Responses in the Link Between Periodontitis and Rheumatoid ArthritisPeriodontal and inflammatory bowel diseases: Is there evidence of complex pathogenic interactions?Lessons learned and unlearned in periodontal microbiologyBenzyl isothiocyanate, a major component from the roots of Salvadora persica is highly active against Gram-negative bacteriaImmunomicrobial pathogenesis of periodontitis: keystones, pathobionts, and host responseDose-response met-RANTES treatment of experimental periodontitis: a narrow edge between the disease severity attenuation and infection control.Baicalin downregulates Porphyromonas gingivalis lipopolysaccharide-upregulated IL-6 and IL-8 expression in human oral keratinocytes by negative regulation of TLR signaling.The Kampo medicine Rokumigan possesses antibiofilm, anti-inflammatory, and wound healing properties.Smoking-related cotinine levels and host responses in chronic periodontitis.The novel role of HtrA1 in gingivitis, chronic and aggressive periodontitis.Divergence of the systemic immune response following oral infection with distinct strains of Porphyromonas gingivalis.Regulation of osteoclast homeostasis and inflammatory bone loss by MFG-E8.Simultaneous analysis of T helper subsets (Th1, Th2, Th9, Th17, Th22, Tfh, Tr1 and Tregs) markers expression in periapical lesions reveals multiple cytokine clusters accountable for lesions activity and inactivity status.High-throughput DNA analysis shows the importance of methylation in the control of immune inflammatory gene transcription in chronic periodontitis.Interactions of adiponectin and lipopolysaccharide from Porphyromonas gingivalis on human oral epithelial cells.Licorice and its potential beneficial effects in common oro-dental diseases.Effects of aging on endotoxin tolerance induced by lipopolysaccharides derived from Porphyromonas gingivalis and Escherichia coli.General immune status and oral microbiology in patients with different forms of periodontitis and healthy control subjects.The plant coumarins auraptene and lacinartin as potential multifunctional therapeutic agents for treating periodontal disease.Aggressive periodontitis defined by recursive partitioning analysis of immunologic factors.Restoring host-microbe homeostasis via selective chemoattraction of Tregs.Macrophage subset sensitivity to endotoxin tolerisation by Porphyromonas gingivalisHyperlipidemia impaired innate immune response to periodontal pathogen porphyromonas gingivalis in apolipoprotein E knockout mice.Glycated albumin produced in diabetic hyperglycemia promotes monocyte secretion of inflammatory cytokines and bacterial adherence to epithelial cells.Current understanding of the relationship between periodontal and systemic diseases.Leukocyte inclusion within a platelet rich plasma-derived fibrin scaffold stimulates a more pro-inflammatory environment and alters fibrin propertiesFOXO1 deletion reduces dendritic cell function and enhances susceptibility to periodontitis.Identification of transactivation-responsive DNA-binding protein 43 (TARDBP43; TDP-43) as a novel factor for TNF-α expression upon lipopolysaccharide stimulation in human monocytes.Aggregatibacter actinomycetemcomitans leukotoxin: a powerful tool with capacity to cause imbalance in the host inflammatory responseTumor Necrosis Factor-α, Matrix-Metalloproteinases 8 and 9 Levels in the Saliva Are Associated with Increased Hemoglobin A1c in Type 1 Diabetes SubjectsIntramembranous bone healing process subsequent to tooth extraction in mice: micro-computed tomography, histomorphometric and molecular characterization.Influence of type 2 diabetes on local production of inflammatory molecules in adults with and without chronic periodontitis: a cross-sectional studyDiabetes aggravates periodontitis by limiting repair through enhanced inflammation.The Daiokanzoto (TJ-84) Kampo Formulation Reduces Virulence Factor Gene Expression in Porphyromonas gingivalis and Possesses Anti-Inflammatory and Anti-Protease Activities.The B Cell-Stimulatory Cytokines BLyS and APRIL Are Elevated in Human Periodontitis and Are Required for B Cell-Dependent Bone Loss in Experimental Murine Periodontitis.Mini but mighty: microRNAs in the pathobiology of periodontal disease.Porphyromonas gingivalis Stimulates Bone Resorption by Enhancing RANKL (Receptor Activator of NF-κB Ligand) through Activation of Toll-like Receptor 2 in Osteoblasts.Oral inflammatory diseases and systemic inflammation: role of the macrophage.IL-4/CCL22/CCR4 axis controls regulatory T-cell migration that suppresses inflammatory bone loss in murine experimental periodontitis
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P2860
Destructive and protective roles of cytokines in periodontitis: a re-appraisal from host defense and tissue destruction viewpoints.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 25 August 2010
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Destructive and protective rol ...... tissue destruction viewpoints.
@en
Destructive and protective rol ...... tissue destruction viewpoints.
@nl
type
label
Destructive and protective rol ...... tissue destruction viewpoints.
@en
Destructive and protective rol ...... tissue destruction viewpoints.
@nl
prefLabel
Destructive and protective rol ...... tissue destruction viewpoints.
@en
Destructive and protective rol ...... tissue destruction viewpoints.
@nl
P356
P1476
Destructive and protective rol ...... tissue destruction viewpoints.
@en
P2093
G P Garlet
P304
P356
10.1177/0022034510376402
P407
P577
2010-08-25T00:00:00Z