about
Selective protein arylation and acetaminophen-induced hepatotoxicity.The ADP-ribosylation domain of Pseudomonas aeruginosa ExoS is required for membrane bleb niche formation and bacterial survival within epithelial cells.Comparative analysis of different methodological approaches to the in vitro study of drug-induced apoptosisTRPC channels and their implication in neurological diseasesThe Impact of Sterile Inflammation in Acute Liver Injury.Hypoxic stress, hepatocytes and CACO-2 viability and susceptibility to Shigella flexneri invasionMitochondrial and extramitochondrial apoptotic signaling pathways in cerebrocortical neurons.Intracellular Ca2+ thresholds that determine survival or death of energy-deprived cells.Cytosolic-free calcium increases to greater than 100 micromolar in ATP-depleted proximal tubules.Pharmacological strategies against cold ischemia reperfusion injury.Continuous bioluminescent monitoring of cytoplasmic ATP in single isolated rat hepatocytes during metabolic poisoning.Normal and abnormal calcium homeostasis in neurons: a basis for the pathophysiology of traumatic and ischemic central nervous system injury.Biochemical basis of hepatocellular injury.Selective inhibition of adenylate cyclase in bovine cortex by quinones: a novel cellular substrate for quinone cytotoxicity.Calcium-dependent opening of a non-specific pore in the mitochondrial inner membrane is inhibited at pH values below 7. Implications for the protective effect of low pH against chemical and hypoxic cell damage.Cytosolic calcium increase in coronary endothelial cells after H2O2 exposure and the inhibitory effect of U78517FChanges in membrane properties during energy depletion-induced cell injury studied with fluorescence microscopy.Intracellular pH response to anoxia in acutely dissociated adult rat hippocampal CA1 neurons.Analysis of Multivariables during Porcine Liver Digestion to Improve Hepatocyte Yield and Viability for Use in Bioartificial Liver Support Systems.The unique histidine in OSCP subunit of F-ATP synthase mediates inhibition of the permeability transition pore by acidic pH.The brain at high altitude: hypometabolism as a defense against chronic hypoxia?TRPC Channels and Parkinson's Disease.Alterations in reactive oxygen, pH, and calcium in astrocytoma cells during lethal injury.
P2860
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 1990
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Calcium and pH in anoxic and toxic injury.
@en
Calcium and pH in anoxic and toxic injury.
@nl
type
label
Calcium and pH in anoxic and toxic injury.
@en
Calcium and pH in anoxic and toxic injury.
@nl
prefLabel
Calcium and pH in anoxic and toxic injury.
@en
Calcium and pH in anoxic and toxic injury.
@nl
P2093
P2860
P1476
Calcium and pH in anoxic and toxic injury.
@en
P2093
P2860
P304
P356
10.3109/10408449009089876
P407
P577
1990-01-01T00:00:00Z