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Do we have to kill the last CML cell?

Do we have to kill the last CML cell?

http://www.wikidata.org/entity/Q37789148

about

Chronic myeloid leukemia stem cells in the era of targeted therapies: resistance, persistence and long-term dormancy.Operational cures after interferon-alpha in patients with chronic myeloid leukemia in Central and Northern Moravia The evolving concept of cancer and metastasis stem cells Genomic instability may originate from imatinib-refractory chronic myeloid leukemia stem cells.Leukemic stem cell persistence in chronic myeloid leukemia patients with sustained undetectable molecular residual disease In search of CML stem cells' deadly weakness.Antigens in chronic myeloid leukemia: implications for vaccine development.Hematopoietic stem cells, progenitor cells and leukemic stem cells in adult myeloproliferative neoplasms.Selection pressure exerted by imatinib therapy leads to disparate outcomes of imatinib discontinuation trials.Dynamics of chronic myeloid leukemia response to long-term targeted therapy reveal treatment effects on leukemic stem cells.Multiple clonal MLL fusions in a patient receiving CHOP-based chemotherapy.Pharmacology and pharmacokinetics of imatinib in pediatric patients.

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Do we have to kill the last CML cell?

http://www.wikidata.org/entity/Q37789148

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 16 September 2010

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Do we have to kill the last CML cell?

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Do we have to kill the last CML cell?

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Do we have to kill the last CML cell?

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Do we have to kill the last CML cell?

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Do we have to kill the last CML cell?

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Do we have to kill the last CML cell?

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J V Melo

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T P Hughes

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P2860

Up-regulation of WRN and DNA ligase IIIalpha in chronic myeloid leukemia: consequences for the repair of DNA double-strand breaks

BCR/ABL induces chromosomal instability after genotoxic stress and alters the cell death threshold

Granulocyte-macrophage progenitors as candidate leukemic stem cells in blast-crisis CML

Imatinib mesylate (STI571) is a substrate for the breast cancer resistance protein (BCRP)/ABCG2 drug pump.

A phase 2 study of imatinib in patients with relapsed or refractory Philadelphia chromosome-positive acute lymphoid leukemias.

Imatinib compared with interferon and low-dose cytarabine for newly diagnosed chronic-phase chronic myeloid leukemia.

Active transport of imatinib into and out of cells: implications for drug resistance.

Effect of a p210 multipeptide vaccine associated with imatinib or interferon in patients with chronic myeloid leukaemia and persistent residual disease: a multicentre observational trial.

Chronic myeloid leukemia and interferon-alpha: a study of complete cytogenetic responders.

Chronic myeloid leukaemia as a model of disease evolution in human cancer.

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193-200

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scholarly article

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10.1038/LEU.2010.197

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2

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2010-09-16T00:00:00Z

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