Endothelial dysfunction as a potential contributor in diabetic nephropathy.
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Nitric oxide synthase derangements and hypertension in kidney diseaseMidkine and the kidney: health and diseasesMidkine in nephrogenesis, hypertension and kidney diseasesRenal endothelial dysfunction in diabetic nephropathyEvaluation of the Zucker diabetic fatty (ZDF) rat as a model for human disease based on urinary peptidomic profilesUric Acid is independently associated with diabetic kidney disease: a cross-sectional study in a Chinese populationDistinct renal pathology and a chemotactic phenotype after enterohemorrhagic Escherichia coli shiga toxins in non-human primate models of hemolytic uremic syndrome.Obesity as an effect modifier of the association between leptin and diabetic kidney disease.Pleiotropic effects of the dipeptidylpeptidase-4 inhibitors on the cardiovascular systemGlobal renal gene expression profiling analysis in B2-kinin receptor null mice: impact of diabetesBlocking αVβ3 integrin ligand occupancy inhibits the progression of albuminuria in diabetic rats.Autophagy in diabetic nephropathyEndothelial dysfunction exacerbates renal interstitial fibrosis through enhancing fibroblast Smad3 linker phosphorylation in the mouse obstructed kidney.Elevated serum levels of mannose-binding lectin and diabetic nephropathy in type 2 diabetes.Proximal tubule dysfunction is associated with podocyte damage biomarkers nephrin and vascular endothelial growth factor in type 2 diabetes mellitus patients: a cross-sectional study.Cytochrome P450 epoxygenase CYP2J2 attenuates nephropathy in streptozotocin-induced diabetic miceFetoplacental vascular endothelial dysfunction as an early phenomenon in the programming of human adult diseases in subjects born from gestational diabetes mellitus or obesity in pregnancy.Nicorandil prevents endothelial dysfunction due to antioxidative effects via normalisation of NADPH oxidase and nitric oxide synthase in streptozotocin diabetic rats.Osmolarity and glucose differentially regulate aldose reductase activity in cultured mouse podocytes.Sirtuin 1: A Target for Kidney Diseases.Endothelial nitric oxide and 15-lipoxygenase-1 metabolites independently mediate relaxation of the rabbit aorta.Arginase inhibition: a new treatment for preventing progression of established diabetic nephropathyEndothelial cell and podocyte autophagy synergistically protect from diabetes-induced glomerulosclerosis.ATP synthase subunit-β down-regulation aggravates diabetic nephropathy.Twenty years after ACEIs and ARBs: emerging treatment strategies for diabetic nephropathy.Sirtuins and renal diseases: relationship with aging and diabetic nephropathy.Hyperfiltration and effect of nitric oxide inhibition on renal and endothelial function in humans with uncomplicated type 1 diabetes mellitus.Protective effects of GLP-1 on glomerular endothelium and its inhibition by PKCβ activation in diabetesMechanisms of bradykinin-induced expression of connective tissue growth factor and nephrin in podocytes.Resting Heart Rate Does Not Predict Cardiovascular and Renal Outcomes in Type 2 Diabetic Patients.Resting heart rate and the risk of microvascular complications in patients with type 2 diabetes mellitusNovel application of a multiscale entropy index as a sensitive tool for detecting subtle vascular abnormalities in the aged and diabetic.Acute Effects of Hemodiafiltration Versus Conventional Hemodialysis on Endothelial Function and Inflammation: A Randomized Crossover Study.Association between fish consumption and nephropathy in American Indians--the Strong Heart Study.Arginase inhibition mediates renal tissue protection in diabetic nephropathy by a nitric oxide synthase 3-dependent mechanismGenetic Targeting of Arginase-II in Mouse Prevents Renal Oxidative Stress and Inflammation in Diet-Induced Obesity.Diabetes-Induced Reactive Oxygen Species: Mechanism of Their Generation and Role in Renal InjuryThe link between metabolic abnormalities and endothelial dysfunction in type 2 diabetes: an update.The renal endothelium in diabetic nephropathy.Cell biology of diabetic nephropathy: Roles of endothelial cells, tubulointerstitial cells and podocytes
P2860
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P2860
Endothelial dysfunction as a potential contributor in diabetic nephropathy.
description
2010 nî lūn-bûn
@nan
2010年の論文
@ja
2010年学术文章
@wuu
2010年学术文章
@zh-cn
2010年学术文章
@zh-hans
2010年学术文章
@zh-my
2010年学术文章
@zh-sg
2010年學術文章
@yue
2010年學術文章
@zh
2010年學術文章
@zh-hant
name
Endothelial dysfunction as a potential contributor in diabetic nephropathy.
@en
Endothelial dysfunction as a potential contributor in diabetic nephropathy.
@nl
type
label
Endothelial dysfunction as a potential contributor in diabetic nephropathy.
@en
Endothelial dysfunction as a potential contributor in diabetic nephropathy.
@nl
prefLabel
Endothelial dysfunction as a potential contributor in diabetic nephropathy.
@en
Endothelial dysfunction as a potential contributor in diabetic nephropathy.
@nl
P2093
P2860
P356
P1476
Endothelial dysfunction as a potential contributor in diabetic nephropathy.
@en
P2093
Byron P Croker
Katsuyuki Tanabe
Maria B Grant
Qiuhong Li
Richard J Johnson
Takahiko Nakagawa
Tomoki Kosugi
P2860
P2888
P356
10.1038/NRNEPH.2010.152
P407
P577
2010-11-02T00:00:00Z
P5875
P6179
1013636625