FLT3 inhibitors in acute myeloid leukemia. - Wikidata - wikimedia - TriplyDB

FLT3 inhibitors in acute myeloid leukemia.

FLT3 inhibitors in acute myeloid leukemia.

http://www.wikidata.org/entity/Q37810061

about

Fluvastatin inhibits FLT3 glycosylation in human and murine cells and prolongs survival of mice with FLT3/ITD leukemia.Mutations of FLT3/ITD confer resistance to multiple tyrosine kinase inhibitors.FLT3 activating mutations display differential sensitivity to multiple tyrosine kinase inhibitors.

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FLT3 inhibitors in acute myeloid leukemia.

http://www.wikidata.org/entity/Q37810061

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on December 2008

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

FLT3 inhibitors in acute myeloid leukemia.

@en

FLT3 inhibitors in acute myeloid leukemia.

@nl

type

label

FLT3 inhibitors in acute myeloid leukemia.

@en

FLT3 inhibitors in acute myeloid leukemia.

@nl

prefLabel

FLT3 inhibitors in acute myeloid leukemia.

@en

FLT3 inhibitors in acute myeloid leukemia.

@nl

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17474086.1.2.153

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Expert Review of Hematology

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FLT3 inhibitors in acute myeloid leukemia.

@en

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B Douglas Smith

http://www.w3.org/2001/XMLSchema#string

Khaled el-Shami

http://www.w3.org/2001/XMLSchema#string

Richard M Stone

http://www.w3.org/2001/XMLSchema#string

P2860

STK-1, the human homolog of Flk-2/Flt-3, is selectively expressed in CD34+ human bone marrow cells and is involved in the proliferation of early progenitor/stem cells

Ligand for FLT3/FLK2 receptor tyrosine kinase regulates growth of haematopoietic stem cells and is encoded by variant RNAs

Activating mutation of D835 within the activation loop of FLT3 in human hematologic malignancies.

A tyrosine kinase created by fusion of the PDGFRA and FIP1L1 genes as a therapeutic target of imatinib in idiopathic hypereosinophilic syndrome.

Single-agent CEP-701, a novel FLT3 inhibitor, shows biologic and clinical activity in patients with relapsed or refractory acute myeloid leukemia.

The impact of FLT3 internal tandem duplication mutant level, number, size, and interaction with NPM1 mutations in a large cohort of young adult patients with acute myeloid leukemia.

Prognostic relevance of FLT3-TKD mutations in AML: the combination matters--an analysis of 3082 patients.

Cell signaling by receptor tyrosine kinases

SHC and SHIP phosphorylation and interaction in response to activation of the FLT3 receptor

A FLT3-targeted tyrosine kinase inhibitor is cytotoxic to leukemia cells in vitro and in vivo

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153-160

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scholarly article

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10.1586/17474086.1.2.153

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2

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1

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2008-12-01T00:00:00Z

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21082920

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