about
Inhibitors of the Hepatitis C Virus Polymerase; Mode of Action and ResistanceThe Evolutionary Dynamics of a Rapidly Mutating Virus within and between Hosts: The Case of Hepatitis C VirusPer-event probability of hepatitis C infection during sharing of injecting equipmentViral quasispecies assembly via maximal clique enumerationSequential bottlenecks drive viral evolution in early acute hepatitis C virus infection.Using approximate Bayesian computation to estimate tuberculosis transmission parameters from genotype data.Incidence of primary hepatitis C infection and risk factors for transmission in an Australian prisoner cohortEvidence that hepatitis C virus genome partly controls infection outcome.A bioinformatics pipeline for the analyses of viral escape dynamics and host immune responses during an infectionGemSIM: general, error-model based simulator of next-generation sequencing data.Accurate single nucleotide variant detection in viral populations by combining probabilistic clustering with a statistical test of strand biasThe epidemiological fitness cost of drug resistance in Mycobacterium tuberculosis.Naturally occurring dominant drug resistance mutations occur infrequently in the setting of recently acquired hepatitis C.Transmitted/Founder Viruses Rapidly Escape from CD8+ T Cell Responses in Acute Hepatitis C Virus InfectionContribution of intra- and interhost dynamics to norovirus evolution.HIV infection and hepatitis C virus genotype 1a are associated with phylogenetic clustering among people with recently acquired hepatitis C virus infection.Current understanding on the role of standard and immunoproteasomes in inflammatory/immunological pathways of multiple sclerosis.Epidemiological and clinical consequences of within-host evolution.Next generation deep sequencing and vaccine design: today and tomorrow.Resistance to hepatitis C virus: potential genetic and immunological determinants.Increasing viral dose causes a reversal in CD8+ T cell immunodominance during primary influenza infection due to differences in antigen presentation, T cell avidity, and precursor numbers.Effects and interactions in an environmentally relevant mixture of pharmaceuticals.A mathematical model of protein degradation by the proteasome.A prospective study of hepatitis C incidence in Australian prisoners.Exploration of genetically determined resistance against hepatitis C infection in high-risk injecting drug users.Short duration of lamivudine for the prevention of hepatitis B virus transmission in pregnancy: lack of potency and selection of resistance mutations.Trade-offs in resource allocation in the intracellular life-cycle of hepatitis C virus.Modeling the in vitro 20S proteasome activity: the effect of PA28-alphabeta and of the sequence and length of polypeptides on the degradation kinetics.spolTools: online utilities for analyzing spoligotypes of the Mycobacterium tuberculosis complex.Single nucleotide polymorphism array profiling identifies distinct chromosomal aberration patterns across colorectal adenomas and carcinomas.A stochastic model for CD8(+)T cell dynamics in human immunosenescence: implications for survival and longevityInterpreting genotype cluster sizes of Mycobacterium tuberculosis isolates typed with IS6110 and spoligotypingExploring and analysing single cell multi-omics data with VDJView
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P50
description
hulumtues
@sq
onderzoeker
@nl
researcher
@en
հետազոտող
@hy
name
Fabio Luciani
@ast
Fabio Luciani
@en
Fabio Luciani
@es
Fabio Luciani
@nl
Fabio Luciani
@sl
type
label
Fabio Luciani
@ast
Fabio Luciani
@en
Fabio Luciani
@es
Fabio Luciani
@nl
Fabio Luciani
@sl
prefLabel
Fabio Luciani
@ast
Fabio Luciani
@en
Fabio Luciani
@es
Fabio Luciani
@nl
Fabio Luciani
@sl
P106
P1153
14050582300
P21
P31
P496
0000-0003-0666-6324