about
Identification and characterization of a soluble form of the plasma cell membrane glycoprotein PC-1 (5'-nucleotide phosphodiesterase)Molecular pathogenesis of infections caused by Legionella pneumophilaInhibition of breast cancer resistance protein (ABCG2) in human myeloid dendritic cells induces potent tolerogenic functions during LPS stimulationA convenient model of severe, high incidence autoimmune gastritis caused by polyclonal effector T cells and without perturbation of regulatory T cells.A method for quantifying pulmonary Legionella pneumophila infection in mouse lungs by flow cytometry.CD4+ CD25+ regulatory T cells modulate the T-cell and antibody responses in helicobacter-infected BALB/c mice.Immune Control of Legionella Infection: An in vivo Perspective.Pharmacological profiles of the murine gastric and colonic H,K-ATPases.Immunopathogenesis, loss of T cell tolerance and genetics of autoimmune gastritis.The Golgi apparatus in the endomembrane-rich gastric parietal cells exist as functional stable mini-stacks dispersed throughout the cytoplasmGenetic control of susceptibility to autoimmune gastritis.Shaping the T cell repertoire to a bona fide autoantigen: lessons from autoimmune gastritis.Influence of gastric acid on susceptibility to infection with ingested bacterial pathogensImpact of glycans on T-cell tolerance to glycosylated self-antigens.Shaping the T-cell repertoire in the periphery.Species-specific distribution of alpha-galactosyl epitopes on the gastric H/K ATPase beta-subunit: relevance to the binding of human anti-parietal cell autoantibodies.Alpha and beta subunits of the gastric H+/K(+)-ATPase are concordantly targeted by parietal cell autoantibodies associated with autoimmune gastritis.IL-11 is a parietal cell cytokine that induces atrophic gastritisLoss of RegI in conjunction with gastrin deficiency in mice facilitates efficient gastric ulcer healing but is dispensable for hyperplasia and tumourigenesis.Both IFN-γ and IL-17 are required for the development of severe autoimmune gastritis.Isolation, culture and adenoviral transduction of parietal cells from mouse gastric mucosa.Transient systemic inflammation does not alter the induction of tolerance to gastric autoantigens by migratory dendritic cells.Gastric parietal cell acid secretion in mice can be regulated independently of H/K ATPase endocytosis.Independent trafficking of the KCNQ1 K+ channel and H+-K+-ATPase in gastric parietal cells from mice.Reduced effects of amphetamine on prepulse inhibition of startle in gastrin-deficient mice.Growth factors associated with gastric mucosal hypertrophy in autoimmune gastritis.CD4+ T cells, but not CD8+ T cells, are required for the development of experimental autoimmune gastritis.Pathogenic T cells persist after reversal of autoimmune disease by immunosuppression with regulatory T cells.Identification of the Gasa3 and Gasa4 autoimmune gastritis susceptibility genes using congenic mice and partitioned, segregative and interaction analyses.CD8 subunit expression by plasmacytoid dendritic cells is variable, and does not define stable subsets.The interleukin-6 family cytokine interleukin-11 regulates homeostatic epithelial cell turnover and promotes gastric tumor development.Sculpting the immune response to infectionThe role of CD4+ CD25+ immunoregulatory T cells in the induction of autoimmune gastritisRequirements for autoimmune responses to mouse gastric autoantigensCutting Edge: Pulmonary Legionella pneumophila Is Controlled by Plasmacytoid Dendritic Cells but Not Type I IFNA microbore high-performance liquid chromatography strategy for the purification of polypeptides for gas-phase sequence analysis. Structural studies on the murine transferrin receptorTwo genetic loci independently confer susceptibility to autoimmune gastritisThree day neonatal thymectomy selectively depletes NK1.1+ T cellsReciprocal changes in trefoil 1 and 2 expression in stomachs of mice with gastric unit hypertrophy and inflammationThe unique ultrastructure of secretory membranes in gastric parietal cells depends upon the presence of H + , K + -ATPase
P50
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P50
description
hulumtues
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researcher
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wetenschapper
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հետազոտող
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name
Ian R van Driel
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Ian R van Driel
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Ian R van Driel
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Ian R van Driel
@nl
Ian R van Driel
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Ian R van Driel
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Ian R van Driel
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Ian R van Driel
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Ian R van Driel
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Ian R van Driel
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prefLabel
Ian R van Driel
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Ian R van Driel
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Ian R van Driel
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Ian R van Driel
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Ian R van Driel
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P106
P1153
7003394803
P21
P31
P496
0000-0001-5371-120X