Evidence of the selective alteration of anthracycline activity due to modulation by ICRF-187 (ADR-529).
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Anthracyclines and the heart.Monohydroxyethylrutoside as protector against chronic doxorubicin-induced cardiotoxicityA comparative study between catalase gene therapy and the cardioprotector monohydroxyethylrutoside (MonoHER) in protecting against doxorubicin-induced cardiotoxicity in vitroPreventing the cardiotoxicity of anthracyclines by dexrazoxaneThe protective effect of cardiac gene transfer of CuZn-sod in comparison with the cardioprotector monohydroxyethylrutoside against doxorubicin-induced cardiotoxicity in cultured cells.
P2860
Evidence of the selective alteration of anthracycline activity due to modulation by ICRF-187 (ADR-529).
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on January 1990
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Evidence of the selective alte ...... ulation by ICRF-187 (ADR-529).
@en
Evidence of the selective alte ...... due to modulation by ICRF-187
@nl
type
label
Evidence of the selective alte ...... ulation by ICRF-187 (ADR-529).
@en
Evidence of the selective alte ...... due to modulation by ICRF-187
@nl
prefLabel
Evidence of the selective alte ...... ulation by ICRF-187 (ADR-529).
@en
Evidence of the selective alte ...... due to modulation by ICRF-187
@nl
P2093
P1476
Evidence of the selective alte ...... ulation by ICRF-187 (ADR-529).
@en
P2093
P356
10.1016/0163-7258(90)90018-W
P577
1990-01-01T00:00:00Z