Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
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A SEL1L mutation links a canine progressive early-onset cerebellar ataxia to the endoplasmic reticulum-associated protein degradation (ERAD) machineryThe Unfolded Protein Response and the Role of Protein Disulfide Isomerase in NeurodegenerationProtein disulfide isomerases in neurodegeneration: from disease mechanisms to biomedical applicationsSurveillance, phagocytosis, and inflammation: how never-resting microglia influence adult hippocampal neurogenesisThe delicate balance between secreted protein folding and endoplasmic reticulum-associated degradation in human physiologyFunctional contribution of the transcription factor ATF4 to the pathogenesis of amyotrophic lateral sclerosisRecent advances in understanding vitiligoMutation of the BiP/GRP78 gene causes axon outgrowth and fasciculation defects in the thalamocortical connections of the mammalian forebrainLoss of function of the ALS protein SigR1 leads to ER pathology associated with defective autophagy and lipid raft disturbancesCharacterization of the structural and functional determinants of MANF/CDNF in Drosophila in vivo modelEndoplasmic reticulum stress and inflammation in the central nervous systemEdaravone improves spatial memory and modulates endoplasmic reticulum stress-mediated apoptosis after abdominal surgery in miceProtein homeostasis of a metastable subproteome associated with Alzheimer's disease.Assessment of common variability and expression quantitative trait loci for genome-wide associations for progressive supranuclear palsy.Guanabenz, which enhances the unfolded protein response, ameliorates mutant SOD1-induced amyotrophic lateral sclerosis.Acute inducible ablation of GRP78 reveals its role in hematopoietic stem cell survival, lymphogenesis and regulation of stress signaling.Protein Disulfide Isomerase Superfamily in Disease and the Regulation of ApoptosisC/EBP homologous protein (CHOP) deficiency aggravates hippocampal cell apoptosis and impairs memory performanceAutophagy impairment: a crossroad between neurodegeneration and tauopathiesSevoflurane induces endoplasmic reticulum stress mediated apoptosis in hippocampal neurons of aging rats.Inflammation and ER stress downregulate BDH2 expression and dysregulate intracellular iron in macrophages.Protein disulfide isomerase interacts with tau protein and inhibits its fibrillization.The MMACHC proteome: hallmarks of functional cobalamin deficiency in humans.Targeting axonal protein synthesis in neuroregeneration and degeneration.Transgenic neuronal overexpression reveals that stringently regulated p23 expression is critical for coordinated movement in miceTargeting the UPR transcription factor XBP1 protects against Huntington's disease through the regulation of FoxO1 and autophagy.Unfolded protein stress in the endoplasmic reticulum and mitochondria: a role in neurodegenerationProtection of Human Pancreatic Islets from Lipotoxicity by Modulation of the TransloconEndoplasmic Reticulum Stress and Unfolded Protein Response Pathways: Potential for Treating Age-related Retinal DegenerationAn S-opsin knock-in mouse (F81Y) reveals a role for the native ligand 11-cis-retinal in cone opsin biosynthesisThe impact of the unfolded protein response on human diseasePerk-dependent repression of miR-106b-25 cluster is required for ER stress-induced apoptosisFörster resonance energy transfer-based sensor targeting endoplasmic reticulum reveals highly oxidative environment.ER stress inhibits neuronal death by promoting autophagyPACAP Protects Adult Neural Stem Cells from the Neurotoxic Effect of Ketamine Associated with Decreased Apoptosis, ER Stress and mTOR Pathway Activation.Experimental study of the protective effects of SYVN1 against diabetic retinopathyCSB ablation induced apoptosis is mediated by increased endoplasmic reticulum stress responseInherited genetic variants in autism-related CNTNAP2 show perturbed trafficking and ATF6 activationHIV-1 gp120 induces type-1 programmed cell death through ER stress employing IRE1α, JNK and AP-1 pathway.Induced pluripotent stem cell models of progranulin-deficient frontotemporal dementia uncover specific reversible neuronal defects.
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Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 31 January 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
@en
Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
@nl
type
label
Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
@en
Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
@nl
prefLabel
Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
@en
Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
@nl
P2093
P1476
Protein folding stress in neurodegenerative diseases: a glimpse into the ER.
@en
P2093
Claudio Hetz
Laurie H Glimcher
Soledad Matus
P304
P356
10.1016/J.CEB.2011.01.003
P577
2011-01-31T00:00:00Z