Extrusions are phagocytosed and promote Chlamydia survival within macrophages.
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Laser-mediated rupture of chlamydial inclusions triggers pathogen egress and host cell necrosis.Comparison of Murine Cervicovaginal Infection by Chlamydial Strains: Identification of Extrusions Shed In vivoChlamydia trachomatis Cellular Exit Alters Interactions with Host Dendritic CellsControl of cytoskeletal dynamics during cellular responses to pore forming toxins.Impact of Gentamicin Concentration and Exposure Time on Intracellular Yersinia pestis.Chlamydia trachomatis inclusion membrane protein MrcA interacts with the inositol 1,4,5-trisphosphate receptor type 3 (ITPR3) to regulate extrusion formation.
P2860
Extrusions are phagocytosed and promote Chlamydia survival within macrophages.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 14 October 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Extrusions are phagocytosed and promote Chlamydia survival within macrophages.
@en
Extrusions are phagocytosed and promote Chlamydia survival within macrophages.
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type
label
Extrusions are phagocytosed and promote Chlamydia survival within macrophages.
@en
Extrusions are phagocytosed and promote Chlamydia survival within macrophages.
@nl
prefLabel
Extrusions are phagocytosed and promote Chlamydia survival within macrophages.
@en
Extrusions are phagocytosed and promote Chlamydia survival within macrophages.
@nl
P2093
P2860
P356
P1476
Extrusions are phagocytosed and promote Chlamydia survival within macrophages
@en
P2093
Anthony Venida
Meghan Zuck
Tisha Ellis
P2860
P356
10.1111/CMI.12683
P577
2016-11-21T00:00:00Z