High Chlamydia Burden Promotes Tumor Necrosis Factor-Dependent Reactive Arthritis in SKG Mice. - Wikidata - wikimedia - TriplyDB

High Chlamydia Burden Promotes Tumor Necrosis Factor-Dependent Reactive Arthritis in SKG Mice.

High Chlamydia Burden Promotes Tumor Necrosis Factor-Dependent Reactive Arthritis in SKG Mice.

http://www.wikidata.org/entity/Q37842142

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[Update on reactive arthritis].In Vivo and Ex Vivo Imaging Reveals a Long-Lasting Chlamydial Infection in the Mouse Gastrointestinal Tract following Genital Tract Inoculation Epidemiology of Clostridium difficile Infection-Associated Reactive Arthritis in Children: An Underdiagnosed, Potentially Morbid Condition.Recent advances and future directions in understanding and treating Chlamydia-induced reactive arthritis.Causality of Chlamydiae in Arthritis and Spondyloarthritis: a Plea for Increased Translational Research.Pathogenesis of ankylosing spondylitis - recent advances and future directions.Potential risk factors for reactive arthritis and persistence of symptoms at 2 years: a case-control study with longitudinal follow-up.

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High Chlamydia Burden Promotes Tumor Necrosis Factor-Dependent Reactive Arthritis in SKG Mice.

http://www.wikidata.org/entity/Q37842142

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article científic

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article scientifique

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artigo científico

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bilimsel makale

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scientific article published on June 2015

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

High Chlamydia Burden Promotes ...... eactive Arthritis in SKG Mice.

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High Chlamydia Burden Promotes ...... eactive Arthritis in SKG Mice.

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High Chlamydia Burden Promotes ...... eactive Arthritis in SKG Mice.

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High Chlamydia Burden Promotes ...... eactive Arthritis in SKG Mice.

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High Chlamydia Burden Promotes ...... eactive Arthritis in SKG Mice.

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High Chlamydia Burden Promotes ...... eactive Arthritis in SKG Mice.

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Arthritis & Rheumatology

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High Chlamydia Burden Promotes ...... Reactive Arthritis in SKG Mice

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Athan C Baillet

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Connor P O'Meara

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Helen Benham

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Jared Velasco

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John V Forrester

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Linda M Rehaume

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Mariapia A Degli-Esposti

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Marina C G Harvie

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Phillip M Hansbro

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Synovial Chlamydia trachomatis up regulates expression of a panel of genes similar to that transcribed by Mycobacterium tuberculosis during persistent infection

Urethral cytokine and immune responses in Chlamydia trachomatis-infected males.

A MyD88-dependent early IL-17 production protects mice against airway infection with the obligate intracellular pathogen Chlamydia muridarum.

TLR2-dependent induction of IL-10 and Foxp3+ CD25+ CD4+ regulatory T cells prevents effective anti-tumor immunity induced by Pam2 lipopeptides in vivo.

Enhanced virulence of Chlamydia muridarum respiratory infections in the absence of TLR2 activation.

Persistence of Chlamydia trachomatis is induced by ciprofloxacin and ofloxacin in vitro

Effects of azithromycin and rifampin on Chlamydia trachomatis infection in vitro.

Combination antibiotics as a treatment for chronic Chlamydia-induced reactive arthritis: a double-blind, placebo-controlled, prospective trial.

TLR2, but not TLR4, is required for effective host defence against Chlamydia respiratory tract infection in early life.

IL-17A synergizes with IFN-γ to upregulate iNOS and NO production and inhibit chlamydial growth.

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1535-1547

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scholarly article

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10.1002/ART.39041

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6

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67

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Charles W. Armitage

Géraldine Brizard

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2015-06-01T00:00:00Z

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271388226

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