Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
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Hypoxia: The Force that Drives Chronic Kidney DiseaseTargeting the intragraft microenvironment and the development of chronic allograft rejectionChronic renal ischemia in humans: can cell therapy repair the kidney in occlusive renovascular disease?Signalling pathways involved in hypoxia-induced renal fibrosis.Chronic central nervous system expression of HIV-1 Tat leads to accelerated rarefaction of neocortical capillaries and loss of red blood cell velocity heterogeneity.A 3-biomarker-panel predicts renal outcome in patients with proteinuric renal diseases.Mechanism of kidney injury caused by bevacizumab in ratsIron-hepcidin dysmetabolism, anemia and renal hypoxia, inflammation and fibrosis in the remnant kidney rat model.Cells of renin lineage express hypoxia inducible factor 2α following experimental ureteral obstruction.The unfolded protein response regulates an angiogenic response by the kidney epithelium during ischemic stressAddition of endothelial progenitor cells to renal revascularization restores medullary tubular oxygen consumption in swine renal artery stenosisRelation of peritubular capillary features to class of lupus nephritisReduced Microvascular Density in Omental Biopsies of Children with Chronic Kidney DiseaseLow proliferative potential and impaired angiogenesis of cultured rat kidney endothelial cellsBlunted transcriptional response to skeletal muscle ischemia in rats with chronic kidney disease: potential role for impaired ischemia-induced angiogenesis.Physiological Capillary Regression is not Dependent on Reducing VEGF Expression.Renal expression of hypoxia inducible factor-1α in patients with chronic kidney disease: a clinicopathologic study from nephrectomized kidneysHuman mesenchymal stromal cell-derived extracellular vesicles alleviate renal ischemic reperfusion injury and enhance angiogenesis in ratsRenal vascular structure and rarefaction.Key Features of the Intragraft Microenvironment that Determine Long-Term Survival Following TransplantationUpdate on implications and mechanisms of angiogenesis in liver fibrosis.Hypoxia as a key player in the AKI-to-CKD transition.Tubular atrophy in the pathogenesis of chronic kidney disease progression.RNA expression signatures and posttranscriptional regulation in diabetic nephropathy.Novel Biomarkers in the Diagnosis of Chronic Kidney Disease and the Prediction of Its Outcome.Gremlin regulates renal inflammation via the vascular endothelial growth factor receptor 2 pathway.Progression of chronic kidney disease: too much cellular talk causes damage.Identification of urinary exosomal noncoding RNAs as novel biomarkers in chronic kidney disease.Bimodal Pattern of Coronary Microvascular Involvement in Diabetes Mellitus.Evaluation of biomarkers of cell cycle arrest and inflammation in prediction of dialysis or recovery after kidney transplantation.Local CD34-positive capillaries decrease in mouse models of kidney disease associating with the severity of glomerular and tubulointerstitial lesions.Cellular senescence, senescence-associated secretory phenotype, and chronic kidney diseaseChronic Hypobaric Hypoxia Modulates Primary Cilia Differently in Adult and Fetal Ovine Kidneys.Adipose-derived mesenchymal stem cells employed exosomes to attenuate AKI-CKD transition through tubular epithelial cell dependent Sox9 activation.An anatomically unbiased approach for analysis of renal BOLD magnetic resonance images.In Situ Hybridization and Double Immunohistochemistry for the Detection of VEGF-A mRNA and CD34/Collagen IV Proteins in Renal Transplant Biopsies.Vascular endothelial growth factor A is associated with the subsequent development of moderate or severe cardiac allograft vasculopathy in pediatric heart transplant recipients.Renal microRNA- and RNA-profiles in progressive chronic kidney disease.Blood capillary rarefaction and lymphatic capillary neoangiogenesis are key contributors to renal allograft fibrosis in an ACE inhibition rat model.Effects of Erythropoietin Receptor Activity on Angiogenesis, Tubular Injury and Fibrosis in Acute Kidney Injury: A "U-Shaped" Relationship.
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Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 17 February 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
@en
Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
@nl
type
label
Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
@en
Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
@nl
prefLabel
Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
@en
Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
@nl
P2860
P31
P921
P356
P1476
Capillary rarefaction, hypoxia, VEGF and angiogenesis in chronic renal disease.
@en
P275
P2860
P304
P356
10.1093/NDT/GFQ832
P407
P50
P5008
P577
2011-02-17T00:00:00Z
2011-04-01T00:00:00Z