Why does chronic inflammation persist: An unexpected role for fibroblasts.
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Analysis of peripheral blood dendritic cells as a non-invasive tool in the follow-up of patients with chronic hepatitis CMolecular Regulation of Toll-like Receptors in Asthma and COPDEctopic lymphoid follicles: inducible centres for generating antigen-specific immune responses within tissuesUnderstanding Fibroblasts in Order to Comprehend the Osteopathic Treatment of the FasciaParticle disease: biologic mechanisms of periprosthetic osteolysis in total hip arthroplasty.Stromal Fibroblasts in Tertiary Lymphoid Structures: A Novel Target in Chronic InflammationPotential of Cells and Cytokines/Chemokines to Regulate Tertiary Lymphoid Structures in Human DiseasesBioregulatory systems medicine: an innovative approach to integrating the science of molecular networks, inflammation, and systems biology with the patient's autoregulatory capacity?SDF-1 in Mammary Fibroblasts of Bovine with Mastitis Induces EMT and Inflammatory Response of Epithelial CellsModulation of TNF-induced macrophage polarization by synovial fibroblasts.Triggering of the dsRNA sensors TLR3, MDA5, and RIG-I induces CD55 expression in synovial fibroblastsCytosolic phospholipase A2 modulates TLR2 signaling in synoviocytesProlonged tumor necrosis factor α primes fibroblast-like synoviocytes in a gene-specific manner by altering chromatin.Adiponectin exacerbates collagen-induced arthritis via enhancing Th17 response and prompting RANKL expressionDownregulation of caveolin‑1 upregulates the expression of growth factors and regulators in co‑culture of fibroblasts with cancer cellsVocal fold fibroblasts immunoregulate activated macrophage phenotypeGene Expression Profiling of IL-17A-Treated Synovial Fibroblasts from the Human Temporomandibular JointSynergy between Hematopoietic and Radioresistant Stromal Cells Is Required for Autoimmune Manifestations of DNase II-/-IFNaR-/- MiceCharacterisation of fibroblast-like synoviocytes from a murine model of joint inflammation.Novel biphasic role of resolvin D1 on expression of cyclooxygenase-2 in lipopolysaccharide-stimulated lung fibroblasts is partly through PI3K/AKT and ERK2 pathways.Rheumatoid synovial fibroblasts differentiate into distinct subsets in the presence of cytokines and cartilage.Elevated cysteine-rich protein 61 (CCN1) promotes skin aging via upregulation of IL-1β in chronically sun-exposed human skinOsteolysis around total knee arthroplasty: a review of pathogenetic mechanisms.Cytokines in rheumatoid arthritis - shaping the immunological landscape.Factors associated with multiple recurrences of nonspecific orbital inflammation aka orbital pseudotumor.Wound healing - A literature review.Controlling the origins of inflammation with a photoactive lipopeptide immunopotentiator.Tumor necrosis factor α induces sustained signaling and a prolonged and unremitting inflammatory response in rheumatoid arthritis synovial fibroblasts.[Fibroblasts as pathogenic cells in rheumatic inflammation].Tumor Necrosis Factor dynamically regulates the mRNA stabilome in rheumatoid arthritis fibroblast-like synoviocytes.Inflammation and its resolution and the musculoskeletal systemStromal fibroblasts derived from mammary gland of bovine with mastitis display inflammation-specific changes.Targeting the adventitial microenvironment in pulmonary hypertension: A potential approach to therapy that considers epigenetic change.Effects of JWH015 in cytokine secretion in primary human keratinocytes and fibroblasts and its suitability for topical/transdermal delivery.Clinical and symptomatological reflections: the fascial system.Lipopolysaccharide responsiveness in vocal fold fibroblasts.Gingival fibroblast responsiveness is differentially affected by Porphyromonas gingivalis: implications for the pathogenesis of periodontitis.Protective effects of paeonol on inflammatory response in IL-1β-induced human fibroblast-like synoviocytes and rheumatoid arthritis progression via modulating NF-κB pathway.Stromal cell cadherin-11 regulates adipose tissue inflammation and diabetes.Intratendon Delivery of Leukocyte-Poor Platelet-Rich Plasma Improves Healing Compared With Leukocyte-Rich Platelet-Rich Plasma in a Rabbit Achilles Tendinopathy Model.
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Why does chronic inflammation persist: An unexpected role for fibroblasts.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 17 February 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Why does chronic inflammation persist: An unexpected role for fibroblasts.
@en
Why does chronic inflammation persist: An unexpected role for fibroblasts.
@nl
type
label
Why does chronic inflammation persist: An unexpected role for fibroblasts.
@en
Why does chronic inflammation persist: An unexpected role for fibroblasts.
@nl
prefLabel
Why does chronic inflammation persist: An unexpected role for fibroblasts.
@en
Why does chronic inflammation persist: An unexpected role for fibroblasts.
@nl
P2860
P1433
P1476
Why does chronic inflammation persist: An unexpected role for fibroblasts.
@en
P2093
Christopher D Buckley
P2860
P356
10.1016/J.IMLET.2011.02.010
P577
2011-02-17T00:00:00Z