Targeted therapy in GIST: in silico modeling for prediction of resistance.
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Insight into molecular dynamics simulation of BRAF(V600E) and potent novel inhibitors for malignant melanomaGSTT1 copy number gain and ZNF overexpression are predictors of poor response to imatinib in gastrointestinal stromal tumorsETV1, 4 and 5: an oncogenic subfamily of ETS transcription factors.Sorafenib inhibits p38α activity in colorectal cancer cells and synergizes with the DFG-in inhibitor SB202190 to increase apoptotic responseComprehensive Analysis of ETS Family Members in Melanoma by Fluorescence In Situ Hybridization Reveals Recurrent ETV1 Amplification.Update on imatinib for gastrointestinal stromal tumors: duration of treatment.NKp30 isoforms and NKp30 ligands are predictive biomarkers of response to imatinib mesylate in metastatic GIST patients.Molecular dynamics reveal BCR-ABL1 polymutants as a unique mechanism of resistance to PAN-BCR-ABL1 kinase inhibitor therapy.The Role of Mast Cells in Molding the Tumor Microenvironment.Role of genetic and molecular profiling in sarcomas.Smoothened (SMO) receptor mutations dictate resistance to vismodegib in basal cell carcinoma.Identification of peptides with ELAV-like mRNA-stabilizing effect: an integrated in vitro/in silico approach.Are two better than one? A novel double-mutant KIT in GIST that responds to ImatinibThrough the open door: Preferential binding of dasatinib to the active form of BCR-ABL unveiled by in silico experimentsMolecular and functional characterization of a new 3' end KIT juxtamembrane deletion in a duodenal GIST treated with neoadjuvant ImatinibKilling two birds with one stone: a case of GIST and supervening CML.Genetic variation at KIT locus may predispose to melanoma.Spectrum of mutations in gastrointestinal stromal tumor patients - a population-based study from Slovakia.Chemical, pharmacological, and in vitro metabolic stability studies on enantiomerically pure RC-33 compounds: promising neuroprotective agents acting as σ₁ receptor agonists.β-Catenin in desmoid-type fibromatosis: deep insights into the role of T41A and S45F mutations on protein structure and gene expression.Mechanism of Resistance in Gastrointestinal Stromal Tumors.
P2860
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P2860
Targeted therapy in GIST: in silico modeling for prediction of resistance.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on March 2011
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vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Targeted therapy in GIST: in silico modeling for prediction of resistance.
@en
Targeted therapy in GIST: in silico modeling for prediction of resistance.
@nl
type
label
Targeted therapy in GIST: in silico modeling for prediction of resistance.
@en
Targeted therapy in GIST: in silico modeling for prediction of resistance.
@nl
prefLabel
Targeted therapy in GIST: in silico modeling for prediction of resistance.
@en
Targeted therapy in GIST: in silico modeling for prediction of resistance.
@nl
P2860
P50
P356
P1476
Targeted therapy in GIST: in silico modeling for prediction of resistance.
@en
P2093
Silvana Pilotti
P2860
P304
P356
10.1038/NRCLINONC.2011.3
P407
P577
2011-03-01T00:00:00Z