Silencing of CDK5 as potential therapy for Alzheimer's disease.
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p120-catenin is necessary for neuroprotection induced by CDK5 silencing in models of Alzheimer's diseaseProtection after stroke: cellular effectors of neurovascular unit integrity.Molecular mechanisms of environmental enrichment: impairments in Akt/GSK3β, neurotrophin-3 and CREB signaling.Tolfenamic acid reduces tau and CDK5 levels: implications for dementia and tauopathies.The Ambiguous Relationship of Oxidative Stress, Tau Hyperphosphorylation, and Autophagy Dysfunction in Alzheimer's Disease.A truncated peptide from p35, a Cdk5 activator, prevents Alzheimer's disease phenotypes in model mice.Special Issue on "Cdk5 and Brain Disorders": PrologueOxidative Stress and Aberrant Cell Cycle in Alzheimer's Disease Lymphocytes: Diagnostic Prospects.Amyloid beta modulators and neuroprotection in Alzheimer's disease: a critical appraisal.
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Silencing of CDK5 as potential therapy for Alzheimer's disease.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on January 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Silencing of CDK5 as potential therapy for Alzheimer's disease.
@en
Silencing of CDK5 as potential therapy for Alzheimer's disease.
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type
label
Silencing of CDK5 as potential therapy for Alzheimer's disease.
@en
Silencing of CDK5 as potential therapy for Alzheimer's disease.
@nl
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Silencing of CDK5 as potential therapy for Alzheimer's disease.
@en
Silencing of CDK5 as potential therapy for Alzheimer's disease.
@nl
P2093
P2860
P356
P1476
Silencing of CDK5 as potential therapy for Alzheimer's disease.
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Alejandro López-Tobón
Diego Piedrahita
Gloria Patricia Cardona-Gómez
John Fredy Castro-Álvarez
Juan Carlos Gallego-Gómez
Ryan L Boudreau
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P304
P356
10.1515/RNS.2011.015
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2011-01-01T00:00:00Z