RANKL inhibition: a promising novel strategy for breast cancer treatment.
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Notch and Mef2 synergize to promote proliferation and metastasis through JNK signal activation in DrosophilaReceptor activator for nuclear factor-κB ligand signaling promotes progesterone-mediated estrogen-induced mammary carcinogenesis.Aberrant Activation of the RANK Signaling Receptor Induces Murine Salivary Gland Tumors.The biology of progesterone receptor in the normal mammary gland and in breast cancerExamining the relationship between diet-induced acidosis and cancer.Progestogens and risk of breast cancer: a link between bone and breast?RANK expression on breast cancer cells promotes skeletal metastasis.OPG and PgR show similar cohort specific effects as prognostic factors in ER positive breast cancerIncreased breast tissue receptor activator of nuclear factor-κB ligand (RANKL) gene expression is associated with higher mammographic density in premenopausal women.Possible mechanisms of the crosstalk between Langerhans cells and regulatory T cells in extramammary Paget disease by receptor activator of nuclear factor kappa B (RANK) ligand/RANK pathways.
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RANKL inhibition: a promising novel strategy for breast cancer treatment.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on April 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
RANKL inhibition: a promising novel strategy for breast cancer treatment.
@en
RANKL inhibition: a promising novel strategy for breast cancer treatment.
@nl
type
label
RANKL inhibition: a promising novel strategy for breast cancer treatment.
@en
RANKL inhibition: a promising novel strategy for breast cancer treatment.
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prefLabel
RANKL inhibition: a promising novel strategy for breast cancer treatment.
@en
RANKL inhibition: a promising novel strategy for breast cancer treatment.
@nl
P2860
P1476
RANKL inhibition: a promising novel strategy for breast cancer treatment
@en
P2093
Eva González-Suárez
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P2888
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P356
10.1007/S12094-011-0646-5
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2011-04-01T00:00:00Z