Entry of the lymphogranuloma venereum strain of Chlamydia trachomatis into host cells involves cholesterol-rich membrane domains.
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Zipper-like internalization of Dr-positive Escherichia coli by epithelial cells is preceded by an adhesin-induced mobilization of raft-associated molecules in the initial step of adhesion.Manipulation of Host Cholesterol by Obligate Intracellular Bacteria.Outbreaks of Mycobacterium tuberculosis MDR strains differentially induce neutrophil respiratory burst involving lipid rafts, p38 MAPK and Syk.Chlamydial infection of monocytes stimulates IL-1beta secretion through activation of the NLRP3 inflammasomeApoB-containing lipoproteins promote infectivity of chlamydial species in human hepatoma cell lineBacterial colonization of host cells in the absence of cholesterol.RNAi screen of Salmonella invasion shows role of COPI in membrane targeting of cholesterol and Cdc42Differences in Chlamydia trachomatis serovar E growth rate in polarized endometrial and endocervical epithelial cells grown in three-dimensional cultureHuman Genome-Wide RNAi Screen for Host Factors That Facilitate Salmonella Invasion Reveals a Role for Potassium Secretion in Promoting Internalization.Membrane cholesterol: a crucial molecule affecting interactions of microbial pathogens with mammalian cellsRole of lipid rafts in E-cadherin-- and HGF-R/Met--mediated entry of Listeria monocytogenes into host cells.Recent insights into the mechanisms of Chlamydia entry.Chlamydial intracellular survival strategies.Comparison of Chlamydia trachomatis serovar L2 growth in polarized genital epithelial cells grown in three-dimensional culture with non-polarized cellsLipid raft-mediated entry is not required for Chlamydia trachomatis infection of cultured epithelial cells.Hijacking and Use of Host Lipids by Intracellular Pathogens.Chlamydia trachomatis Inclusion Disrupts Host Cell Cytokinesis to Enhance Its Growth in Multinuclear Cells.Modulation of host signaling and cellular responses by ChlamydiaCFTR is required for cellular entry and internalization of Chlamydia trachomatis.Human genetic variation in VAC14 regulates Salmonella invasion and typhoid fever through modulation of cholesterol.Porphyromonas gingivalis entry into gingival epithelial cells modulated by Fusobacterium nucleatum is dependent on lipid rafts.High-polarity Mycobacterium avium-derived lipids interact with murine macrophage lipid rafts.The phagocyte NADPH oxidase depends on cholesterol-enriched membrane microdomains for assembly.Invasion of enteropathogenic Escherichia coli into host cells through epithelial tight junctions.Impact of Dietary Cholesterol on the Pathophysiology of Infectious and Autoimmune Disease.Genetic variation in is associated with bacteremia secondary to diverse pathogens in African children
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Entry of the lymphogranuloma venereum strain of Chlamydia trachomatis into host cells involves cholesterol-rich membrane domains.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
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artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2003
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
@cs
name
Entry of the lymphogranuloma v ...... esterol-rich membrane domains.
@en
Entry of the lymphogranuloma v ...... esterol-rich membrane domains.
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type
label
Entry of the lymphogranuloma v ...... esterol-rich membrane domains.
@en
Entry of the lymphogranuloma v ...... esterol-rich membrane domains.
@nl
prefLabel
Entry of the lymphogranuloma v ...... esterol-rich membrane domains.
@en
Entry of the lymphogranuloma v ...... esterol-rich membrane domains.
@nl
P2860
P1476
Entry of the lymphogranuloma v ...... lesterol-rich membrane domains
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P2093
Alice Dautry-Varsat
Isabelle Jutras
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10.1128/IAI.71.1.260-266.2003
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P577
2003-01-01T00:00:00Z