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Matrix Metalloproteinases and Subclinical Atherosclerosis in Chronic Kidney Disease: A Systematic ReviewCross-talk between endothelin-1 and mineral metabolism in hemodialysis patients: a cross-sectional study.Global metabolomic profiling of acute myocarditis caused by Trypanosoma cruzi infectionp-Cresol affects reactive oxygen species generation, cell cycle arrest, cytotoxicity and inflammation/atherosclerosis-related modulators production in endothelial cells and mononuclear cells.Oxidative DNA damage and mortality in hemodialysis and peritoneal dialysis patients.Uremic serum and solutes increase post-vascular interventional thrombotic risk through altered stability of smooth muscle cell tissue factor.Caffeic acid, a phenol found in white wine, modulates endothelial nitric oxide production and protects from oxidative stress-associated endothelial cell injury.Candidate Gene Analysis of Mortality in Dialysis PatientsSkin Autofluorescence Is Associated with Endothelial Dysfunction in Uremic Subjects on Hemodialysis.Increased Levels of Modified Advanced Oxidation Protein Products Are Associated with Central and Peripheral Blood Pressure in Peritoneal Dialysis Patients.Challenges and opportunities for stem cell therapy in patients with chronic kidney diseaseMobilization of Endothelial Progenitor Cells Following Creation of Arteriovenous Access in Patients with End-Stage Renal Disease.Protein-bound uremic toxins stimulate crosstalk between leukocytes and vessel wall.Paraoxonase 1, atherosclerosis and arterial stiffness in renal patients.Circulating endothelial cells and chronic kidney disease.Vascular damage in kidney disease: beyond hypertension.Hemodynamic Shear Stress and Endothelial Dysfunction in Hemodialysis Access.Role of β2-microglobulin in uremic patients may be greater than originally suspected.Arterial Stiffness: A Novel Risk Factor for Kidney Injury Progression?Future research directions to improve fistula maturation and reduce access failure.Cilostazol inhibits uremic toxin-induced vascular smooth muscle cell dysfunction: role of Axl signaling.The Clinical Role of CT-Based Morphologic Description in Severely Calcified Coronary Arteries Ectasia Encountering Acute Coronary Syndrome.Vasculopathy in the setting of cardiorenal syndrome: roles of protein-bound uremic toxins.Antiproliferation effect of the uremic toxin para‑cresol on endothelial progenitor cells is related to its antioxidant activity.Chronic kidney disease alters vascular smooth muscle cell phenotype.The effects of low-dose nepsilon-(carboxymethyl)lysine (CML) and nepsilon-(carboxyethyl)lysine (CEL), two main glycation free adducts considered as potential uremic toxins, on endothelial progenitor cell function.Dialysis Vascular Access Intervention and the Search for Biomarkers.P-cresol, but not p-cresylsulphate, disrupts endothelial progenitor cell function in vitro.Predicting the Functionality and Form of a Dialysis Fistula.Indoxyl sulfate enhances IL-1β-induced E-selectin expression in endothelial cells in acute kidney injury by the ROS/MAPKs/NFκB/AP-1 pathway.Uremic Advanced Glycation End Products and Protein-Bound Solutes Induce Endothelial Dysfunction Through Suppression of Krüppel-Like Factor 2.CX3CL1/CX3CR1 axis, as The Therapeutic Potential In Renal Diseases: Friend Or Foe?Sequential venous anastomosis design to enhance patency of arterio-venous grafts for hemodialysis.Deleterious effects of phosphate on vascular and endothelial function via disruption to the nitric oxide pathway.Para-cresyl sulfate acutely impairs vascular reactivity and induces vascular remodeling.Serum free indoxyl sulfate associated with in-stent restenosis after coronary artery stentings.Distinct Effects of Inorganic Phosphate on Cell Cycle and Apoptosis in Human Vascular Smooth Muscle Cells.Hemodiafiltration is associated with reduced inflammation, oxidative stress and improved endothelial risk profile compared to high-flux hemodialysis in children.Critical Appraisal of Advanced Glycation End Products (AGEs) and Circulating Soluble Receptors for Advanced Glycation End Products (sRAGE) as a Predictive Biomarkers for Cardiovascular Disease in Hemodialysis Patients.The Impact of Uremic Toxins on Vascular Smooth Muscle Cell Function.
P2860
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 21 June 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Does uremia cause vascular dysfunction?
@en
Does uremia cause vascular dysfunction?
@nl
type
label
Does uremia cause vascular dysfunction?
@en
Does uremia cause vascular dysfunction?
@nl
prefLabel
Does uremia cause vascular dysfunction?
@en
Does uremia cause vascular dysfunction?
@nl
P2093
P2860
P356
P1476
Does uremia cause vascular dysfunction?
@en
P2093
Angel Argiles
Ariane Duval-Sabatier
Bertrand Gondouin
Claire Cerini
Françoise Dignat-George
Laetitia Dou
Noémie Jourde-Chiche
Philippe Brunet
Stéphane Burtey
P2860
P304
P356
10.1159/000327131
P577
2011-06-21T00:00:00Z