Tumor necrosis factor in the pathophysiology of infection and sepsis.
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A(2A) adenosine receptors in human peripheral blood cellsReduced endotoxin-induced production of tumor necrosis factor activity by equine peritoneal macrophages exposed to the dual inhibitor of arachidonic acid metabolism, SK & F 86002Dysregulation of in vitro cytokine production by monocytes during sepsisExtracts of mosquito salivary gland inhibit tumour necrosis factor alpha release from mast cells.Tumor necrosis factor (TNF)-dependent shedding of the p55 TNF receptor in a baboon model of bacteremia.Safety, immunogenicity, and transmissibility in humans of CVD 1203, a live oral Shigella flexneri 2a vaccine candidate attenuated by deletions in aroA and virG.Ca2+ responses to interleukin 1 and tumor necrosis factor in cultured human skin fibroblasts. Possible implications for Reye syndrome.Influence of antimicrobial therapy on kinetics of tumor necrosis factor levels in experimental endocarditis caused by Klebsiella pneumoniaeMortality in Sepsis and its relationship with GenderImmunoregulatory biological response modifiers: effect of cytokines on septic shockCryptococcal polysaccharides induce L-selectin shedding and tumor necrosis factor receptor loss from the surface of human neutrophils.Imaging for infection: from visualization of inflammation to visualization of microbes.R1 autonomic nervous system in acute kidney injury.Consequences of interaction of a lipophilic endotoxin antagonist with plasma lipoproteinsExpression of cytokine and chemokine genes by human middle ear epithelial cells induced by influenza A virus and Streptococcus pneumoniae opacity variants.Noradrenaline inhibits lipopolysaccharide-induced tumor necrosis factor and interleukin 6 production in human whole blood.Apoptosis induced by tumor necrosis factor in cells chronically infected with feline immunodeficiency virus.Pro-inflammatory cytokines in a ruminant model: pathophysiological, pharmacological, and therapeutic aspects.Control of adipocyte differentiation.The inducibility of TNF-alpha production is different in the granulocytic and monocytic differentiated forms of wild type and CGD-mutant PLB-985 cells.Preventive treatment of intranasal fluticasone propionate reduces cytokine mRNA expressing cells before and during a single nasal allergen provocation.Fever and acute phase response induced in dwarf goats by endotoxin and bovine and human recombinant tumour necrosis factor alpha.Endotoxins modulate the autocrine function of organ cultured donor corneas and increase the incidence of endothelial cell death.Immunological responses in patients with tuberculosis and in vivo effects of acetyl-L-carnitine oral administration.Liver failure induces a systemic inflammatory response. Prevention by recombinant N-terminal bactericidal/permeability-increasing proteinTumor necrosis factor-alpha-induced insulin resistance in 3T3-L1 adipocytes is accompanied by a loss of insulin receptor substrate-1 and GLUT4 expression without a loss of insulin receptor-mediated signal transduction.Regulation of interleukin 10 release by tumor necrosis factor in humans and chimpanzees.Effects of in vitro ethanol on tumor necrosis factor-alpha production by blood obtained from simian immunodeficiency virus-infected rhesus macaques.Nicotinamide is a potent inhibitor of proinflammatory cytokines.Interleukin-1 promotes coagulation, which is necessary for protective immunity in the lung against Streptococcus pneumoniae infection.Intravitreal treatment with antisense oligonucleotides targeting tumor necrosis factor-α in murine herpes simplex virus type 1 retinitis.In vitro ethanol suppresses alveolar macrophage TNF-alpha during simian immunodeficiency virus infection.Ependymal cells of the choroid plexus express tumour necrosis factor-alpha.Equivalence of the acute cytokine surge and myocardial injury after coronary artery bypass grafting with and without a novel extracorporeal circulation system.Characterization of calves exhibiting a novel inheritable TNF-alpha hyperresponsiveness to endotoxin: associations with increased pathophysiological complications.Murine tumor cells metastasizing selectively in the liver: ability to produce hepatocyte-activating cytokines interleukin-1 and/or -6.
P2860
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P2860
Tumor necrosis factor in the pathophysiology of infection and sepsis.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
Tumor necrosis factor in the pathophysiology of infection and sepsis.
@en
Tumor necrosis factor in the pathophysiology of infection and sepsis.
@nl
type
label
Tumor necrosis factor in the pathophysiology of infection and sepsis.
@en
Tumor necrosis factor in the pathophysiology of infection and sepsis.
@nl
prefLabel
Tumor necrosis factor in the pathophysiology of infection and sepsis.
@en
Tumor necrosis factor in the pathophysiology of infection and sepsis.
@nl
P1476
Tumor necrosis factor in the pathophysiology of infection and sepsis.
@en
P304
P356
10.1016/0090-1229(90)90094-7
P577
1990-05-01T00:00:00Z