Fibroblast growth factor 23 as a phosphotropic hormone and beyond. - Wikidata - wikimedia - TriplyDB

Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

http://www.wikidata.org/entity/Q37913097

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FGF23 and Phosphate Wasting Disorders Effects of Different 1-34 Parathyroid Hormone Dosages on Fibroblast Growth Factor-23 Secretion in Human Bone Marrow Cells following Osteogenic Differentiation.Current concepts in perinatal mineral metabolism Bone and Muscle Endocrine Functions: Unexpected Paradigms of Inter-organ Communication.What's new in FGF23 research?Hypophosphatemic rickets: unraveling the role of FGF23.FGF23 associated bone diseases.FGF23-FGF Receptor/Klotho Pathway as a New Drug Target for Disorders of Bone and Mineral Metabolism.Phosphate enhances Fgf23 expression through reactive oxygen species in UMR-106 cells.What can FGF23 do without Klotho?Targeting Fibroblast Growth Factor 23 Signaling with Antibodies and Inhibitors, Is There a Rationale?Magnetic Resonance Imaging Features as Surrogate Markers of X-Linked Hypophosphatemic Rickets Activity

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Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

http://www.wikidata.org/entity/Q37913097

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artikel ilmiah

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Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

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Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

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type

label

Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

@en

Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

@nl

prefLabel

Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

@en

Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

@nl

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S00774-011-0298-0

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Journal of Bone and Mineral Metabolism

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Fibroblast growth factor 23 as a phosphotropic hormone and beyond.

@en

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Yuichiro Shimizu

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P2860

Autosomal dominant hypophosphataemic rickets is associated with mutations in FGF23

FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis

Mutation of the mouse klotho gene leads to a syndrome resembling ageing

Cloning and characterization of FGF23 as a causative factor of tumor-induced osteomalacia

Autosomal-dominant hypophosphatemic rickets (ADHR) mutations stabilize FGF-23

Definition, evaluation, and classification of renal osteodystrophy: a position statement from Kidney Disease: Improving Global Outcomes (KDIGO)

Mutations in GALNT3, encoding a protein involved in O-linked glycosylation, cause familial tumoral calcinosis

Klotho converts canonical FGF receptor into a specific receptor for FGF23

Homozygous ablation of fibroblast growth factor-23 results in hyperphosphatemia and impaired skeletogenesis, and reverses hypophosphatemia in Phex-deficient mice

Regulation of fibroblast growth factor-23 signaling by klotho

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s00774-011-0298-0

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507-514

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scholarly article

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10.1007/S00774-011-0298-0

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5

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29

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2011-08-06T00:00:00Z

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21822586

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