Constructing immunoprofiles to deconstruct disease complexity in pemphigus. - Wikidata - wikimedia - TriplyDB

Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

http://www.wikidata.org/entity/Q37933433

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Molecular diagnosis in autoimmune skin blistering conditions Pemphigus vulgaris autoantibody profiling by proteomic technique.Critical Role of the Neonatal Fc Receptor (FcRn) in the Pathogenic Action of Antimitochondrial Autoantibodies Synergizing with Anti-desmoglein Autoantibodies in Pemphigus Vulgaris.Autoimmunity in 2012.Monopathogenic vs multipathogenic explanations of pemphigus pathophysiology.Pemphigus vulgaris autoimmune globulin induces Src-dependent tyrosine-phosphorylation of plakophilin 3 and its detachment from desmoglein 3.Specific immunoglobulin isotypes correlate with disease activity, morphology, duration and HLA association in Pemphigus vulgaris.Detailed profiling of anti-desmoglein autoantibodies identifies anti-Dsg1 reactivity as a key driver of disease activity and clinical expression in pemphigus vulgaris.

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P2860

Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

http://www.wikidata.org/entity/Q37933433

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Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

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Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

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Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

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Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

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Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

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Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

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Constructing immunoprofiles to deconstruct disease complexity in pemphigus.

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Animesh A Sinha

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P2860

Dichotomy of autoreactive Th1 and Th2 cell responses to desmoglein 3 in patients with pemphigus vulgaris (PV) and healthy carriers of PV-associated HLA class II alleles

Autoantibodies against a novel epithelial cadherin in pemphigus vulgaris, a disease of cell adhesion

Loss of the acyl-CoA binding protein (Acbp) results in fatty acid metabolism abnormalities in mouse hair and skin

HLA class II allelic variation and susceptibility to pemphigus vulgaris

Consensus statement on definitions of disease, end points, and therapeutic response for pemphigus.

Structural basis for major histocompatibility complex (MHC)-linked susceptibility to autoimmunity: charged residues of a single MHC binding pocket confer selective presentation of self-peptides in pemphigus vulgaris.

Analysis of the T cells that are potentially involved in autoantibody production in pemphigus vulgaris.

Autoimmunity to keratinocyte acetylcholine receptors in pemphigus.

Disease relevant HLA class II alleles isolated by genotypic, haplotypic, and sequence analysis in North American Caucasians with pemphigus vulgaris.

Analysis of intracellular cytokines using flowcytometry.

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36-43

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10.3109/08916934.2011.606445

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