The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
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Soluble Urokinase Receptors in Focal Segmental Glomerulosclerosis: A Review on the Scientific Point of ViewCalcium, TRPC channels, and regulation of the actin cytoskeleton in podocytes: towards a future of targeted therapiesRoles of adaptor proteins in podocyte biologyMembrane trafficking in podocyte health and diseaseProtective effects of the mTOR inhibitor everolimus on cytoskeletal injury in human podocytes are mediated by RhoA signalingTowards an understanding of kidney diseases associated with WT1 mutationsBiochemical and Cellular Determinants of Renal Glomerular ElasticityStructural Analysis of the Myo1c and Neph1 Complex Provides Insight into the Intracellular Movement of Neph1Fragility of foot process morphology in kidney podocytes arises from chaotic spatial propagation of cytoskeletal instability.Shiga toxin promotes podocyte injury in experimental hemolytic uremic syndrome via activation of the alternative pathway of complementRegulation of Ste20-like kinase, SLK, activity: Dimerization and activation segment phosphorylation.Initial steroid sensitivity in children with steroid-resistant nephrotic syndrome predicts post-transplant recurrenceA comprehensive analysis and annotation of human normal urinary proteome.Glycogen synthase kinase 3β dictates podocyte motility and focal adhesion turnover by modulating paxillin activity: implications for the protective effect of low-dose lithium in podocytopathy.Visualization of cytoskeletal dynamics in podocytes using adenoviral vectors.Ginseng total saponin modulates the changes of α-actinin-4 in podocytes induced by diabetic conditions.A homozygous missense mutation in the ciliary gene TTC21B causes familial FSGSLoss-of-function mutations in WDR73 are responsible for microcephaly and steroid-resistant nephrotic syndrome: Galloway-Mowat syndrome.Trehalose, an mTOR independent autophagy inducer, alleviates human podocyte injury after puromycin aminonucleoside treatment.Endothelin and the podocyteSchip1 is a novel podocyte foot process protein that mediates actin cytoskeleton rearrangements and forms a complex with Nherf2 and ezrin.Fine-tuning of NFκB by glycogen synthase kinase 3β directs the fate of glomerular podocytes upon injury.Renal Lipotoxicity-Associated Inflammation and Insulin Resistance Affects Actin Cytoskeleton Organization in PodocytesmiR-135 family members mediate podocyte injury through the activation of Wnt/β-catenin signaling.The Future of Polycystic Kidney Disease Research--As Seen By the 12 Kaplan Awardees.Calpain-Mediated Cleavage of Calcineurin in Puromycin Aminonucleoside-Induced Podocyte Injury.Anti-TGF-β Antibody, 1D11, Ameliorates Glomerular Fibrosis in Mouse Models after the Onset of Proteinuria.The Prediction of Key Cytoskeleton Components Involved in Glomerular Diseases Based on a Protein-Protein Interaction Network.Functional Validation of an Alpha-Actinin-4 Mutation as a Potential Cause of an Aggressive Presentation of Adolescent Focal Segmental Glomerulosclerosis: Implications for Genetic Testing.MiR-30a Inhibits the Epithelial--Mesenchymal Transition of Podocytes through Downregulation of NFATc3MicroRNA-30 family members regulate calcium/calcineurin signaling in podocytesPodocyte-specific knockout of myosin 1e disrupts glomerular filtrationAutoantibodies targeting glomerular annexin A2 identify patients with proliferative lupus nephritis.Current concepts of the podocyte in nephrotic syndrome.The redox sensitive glycogen synthase kinase 3β suppresses the self-protective antioxidant response in podocytes upon oxidative glomerular injury.Angiotensin II induces endoplasmic reticulum stress in podocyte, which would be further augmented by PI3-kinase inhibitionProstaglandin E2 promotes cellular recovery from established nephrotoxic serum nephritis in mice, prosurvival, and regenerative effects on glomerular cells.Angiotensin II contributes to diabetic renal dysfunction in rodents and humans via Notch1/Snail pathwayGenetic Ablation of Calcium-independent Phospholipase A2γ Induces Glomerular Injury in Mice.Calcineurin inhibitors cyclosporin A and tacrolimus protect against podocyte injury induced by puromycin aminonucleoside in rodent models
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The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
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artikel ilmiah
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artikull shkencor
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artículo científico
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name
The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
@en
The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
@nl
type
label
The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
@en
The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
@nl
prefLabel
The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
@en
The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
@nl
P2860
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P1476
The podocyte cytoskeleton--key to a functioning glomerulus in health and disease.
@en
P2093
Gavin I Welsh
Moin A Saleem
P2860
P2888
P356
10.1038/NRNEPH.2011.151
P407
P577
2011-10-25T00:00:00Z
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P6179
1036838722