Spatial and temporal activation of spinal glial cells: role of gliopathy in central neuropathic pain following spinal cord injury in rats. - Wikidata - wikimedia - TriplyDB

Spatial and temporal activation of spinal glial cells: role of gliopathy in central neuropathic pain following spinal cord injury in rats.

Spatial and temporal activation of spinal glial cells: role of gliopathy in central neuropathic pain following spinal cord injury in rats.

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Pathophysiology, Clinical Importance, and Management of Neurogenic Lower Urinary Tract Dysfunction Caused by Suprasacral Spinal Cord Injury Spinal Gap Junction Channels in Neuropathic Pain Mechanisms of chemotherapy-induced behavioral toxicities Reciprocal modulation between microglia and astrocyte in reactive gliosis following the CNS injury Human stem cell-derived spinal cord astrocytes with defined mature or reactive phenotypes ARA 290, a peptide derived from the tertiary structure of erythropoietin, produces long-term relief of neuropathic pain coupled with suppression of the spinal microglia response Targeting the Microglial Signaling Pathways: New Insights in the Modulation of Neuropathic Pain.Overexpression of the astrocyte glutamate transporter GLT1 exacerbates phrenic motor neuron degeneration, diaphragm compromise, and forelimb motor dysfunction following cervical contusion spinal cord injury.Spinal cord transection-induced allodynia in rats--behavioral, physiopathological and pharmacological characterization Bone marrow mesenchymal stem cells with Nogo-66 receptor gene silencing for repair of spinal cord injury.Chronic at-level thermal hyperalgesia following rat cervical contusion spinal cord injury is accompanied by neuronal and astrocyte activation and loss of the astrocyte glutamate transporter, GLT1, in superficial dorsal horn.Morphine self-administration following spinal cord injury.The generation of definitive neural stem cells from PiggyBac transposon-induced pluripotent stem cells can be enhanced by induction of the NOTCH signaling pathway.Recent advances in the pharmacologic treatment of spinal cord injury Reorganization of the intact somatosensory cortex immediately after spinal cord injury.The major brain endocannabinoid 2-AG controls neuropathic pain and mechanical hyperalgesia in patients with neuromyelitis optica.Transplantation of glial progenitors that overexpress glutamate transporter GLT1 preserves diaphragm function following cervical SCI.Somatosensory phenotype is associated with thalamic metabolites and pain intensity after spinal cord injury Human iPS cell-derived astrocyte transplants preserve respiratory function after spinal cord injury.α7 Nicotinic Receptor Promotes the Neuroprotective Functions of Astrocytes against Oxaliplatin Neurotoxicity Glia as a Link between Neuroinflammation and Neuropathic Pain.Age differences in cytokine expression under conditions of health using experimental pain models.Neuropathic Pain and Spinal Cord Injury: Phenotypes and Pharmacological Management.GLT1 overexpression reverses established neuropathic pain-related behavior and attenuates chronic dorsal horn neuron activation following cervical spinal cord injury.Gabapentin reduces CX3CL1 signaling and blocks spinal microglial activation in monoarthritic rats Sparing of Descending Axons Rescues Interneuron Plasticity in the Lumbar Cord to Allow Adaptive Learning After Thoracic Spinal Cord Injury Neuron-astrocyte signaling network in spinal cord dorsal horn mediates painful neuropathy of type 2 diabetes.Spinal astrocytes produce and secrete dynorphin neuropeptides.Metabolite concentrations in the anterior cingulate cortex predict high neuropathic pain impact after spinal cord injury.Cell cycle inhibition limits development and maintenance of neuropathic pain following spinal cord injury.Cell cycle activation contributes to increased neuronal activity in the posterior thalamic nucleus and associated chronic hyperesthesia after rat spinal cord contusion TrkB.T1 contributes to neuropathic pain after spinal cord injury through regulation of cell cycle pathways.Tissue plasminogen activator contributes to morphine tolerance and induces mechanical allodynia via astrocytic IL-1β and ERK signaling in the spinal cord of mice Recombinant neural progenitor transplants in the spinal dorsal horn alleviate chronic central neuropathic pain.Identification of A3 adenosine receptor agonists as novel non-narcotic analgesics.Thrombospondin-4 contributes to spinal cord injury-induced changes in nociception.Glia and pain: is chronic pain a gliopathy?In vivo direct reprogramming of reactive glial cells into functional neurons after brain injury and in an Alzheimer's disease model.Effect of ZBD-2 on chronic pain, depressive-like behaviors, and recovery of motor function following spinal cord injury in mice.Opioid administration following spinal cord injury: implications for pain and locomotor recovery.

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Spatial and temporal activation of spinal glial cells: role of gliopathy in central neuropathic pain following spinal cord injury in rats.

http://www.wikidata.org/entity/Q37950711

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Spatial and temporal activatio ...... ng spinal cord injury in rats.

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Spatial and temporal activatio ...... ng spinal cord injury in rats.

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Spatial and temporal activatio ...... ng spinal cord injury in rats.

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Spatial and temporal activatio ...... ng spinal cord injury in rats.

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Spatial and temporal activatio ...... ng spinal cord injury in rats.

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Spatial and temporal activatio ...... ng spinal cord injury in rats.

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J.EXPNEUROL.2011.10.010

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Experimental Neurology

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Spatial and temporal activatio ...... ng spinal cord injury in rats.

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Claire E Hulsebosch

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Geda C Unabia

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Jonghoon Kang

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Young S Gwak

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P2860

Quantitative analysis of cellular inflammation after traumatic spinal cord injury: evidence for a multiphasic inflammatory response in the acute to chronic environment

Mechanisms of chronic central neuropathic pain after spinal cord injury

Attenuation of astrogliosis by suppressing of microglial proliferation with the cell cycle inhibitor olomoucine in rat spinal cord injury model.

Astrocytes initiate inflammation in the injured mouse spinal cord by promoting the entry of neutrophils and inflammatory monocytes in an IL-1 receptor/MyD88-dependent fashion.

The LTB4-BLT1 axis mediates neutrophil infiltration and secondary injury in experimental spinal cord injury.

Modulation of thalamic nociceptive processing after spinal cord injury through remote activation of thalamic microglia by cysteine cysteine chemokine ligand 21

A fundamental role for the nitric oxide-G-kinase signaling pathway in mediating intercellular Ca(2+) waves in glia

Olprinone attenuates the acute inflammatory response and apoptosis after spinal cord trauma in mice

Temporal-spatial expressions of p27kip1 and its phosphorylation on Serine-10 after acute spinal cord injury in adult rat: Implications for post-traumatic glial proliferation

Upregulation of sodium channel Nav1.3 and functional involvement in neuronal hyperexcitability associated with central neuropathic pain after spinal cord injury

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362-372

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10.1016/J.EXPNEUROL.2011.10.010

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2011-10-21T00:00:00Z

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51756101

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22036747

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neuropathic pain

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