Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy. - Wikidata - wikimedia - TriplyDB

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

http://www.wikidata.org/entity/Q37951439

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Diabetic cardiomyopathy: bench to bedside Over-nutrition and metabolic cardiomyopathy Heme oxygenase-1 prevents cardiac dysfunction in streptozotocin-diabetic mice by reducing inflammation, oxidative stress, apoptosis and enhancing autophagy Astragalus polysaccharides improve cardiomyopathy in STZ-induced diabetic mice and heterozygous (SOD2+/-) knockout mice.Activation of EGFR/ERBB2 via pathways involving ERK1/2, P38 MAPK, AKT and FOXO enhances recovery of diabetic hearts from ischemia-reperfusion injury Small engine, big power: microRNAs as regulators of cardiac diseases and regeneration.The role of oxidative stress in high glucose-induced apoptosis in neonatal rat cardiomyocytes.The prevention of diabetic cardiomyopathy by non-mitogenic acidic fibroblast growth factor is probably mediated by the suppression of oxidative stress and damage Association of neutrophil/lymphocyte ratio with obstructive coronary artery disease and coronary artery calcium score detected by multislice computed tomography in type 2 diabetes mellitus patients.Mineralocorticoid receptor blockade prevents Western diet-induced diastolic dysfunction in female mice.Sirtuin-6 deficiency exacerbates diabetes-induced impairment of wound healing.α-Lipoic acid attenuates cardiac fibrosis in Otsuka Long-Evans Tokushima Fatty rats.Molecular and metabolic mechanisms of cardiac dysfunction in diabetes.The functional state of hormone-sensitive adenylyl cyclase signaling system in diabetes mellitus Long term liver specific glucokinase gene defect induced diabetic cardiomyopathy by up regulating NADPH oxidase and down regulating insulin receptor and p-AMPK.Contribution of apoptosis in myocardial reperfusion injury and loss of cardioprotection in diabetes mellitus.miR-30c Mediates Upregulation of Cdc42 and Pak1 in Diabetic Cardiomyopathy.Pyrroloquinoline quinone protects mouse brain endothelial cells from high glucose-induced damage in vitro.Combination of Morroniside and Diosgenin Prevents High Glucose-Induced Cardiomyocytes Apoptosis.Matrine pretreatment improves cardiac function in rats with diabetic cardiomyopathy via suppressing ROS/TLR-4 signaling pathway.Diabetes mellitus activates fetal gene program and intensifies cardiac remodeling and oxidative stress in aged spontaneously hypertensive rats.Losartan reduces myocardial interstitial fibrosis in diabetic cardiomyopathy rats by inhibiting JAK/STAT signaling pathway.Low Intensity Physical Exercise Attenuates Cardiac Remodeling and Myocardial Oxidative Stress and Dysfunction in Diabetic Rats.Iron toxicity mediated by oxidative stress enhances tissue damage in an animal model of diabetes.Alterations of relaxin and its receptor system components in experimental diabetic cardiomyopathy rats.Epigallocatechin-3-gallate combined with alpha lipoic acid attenuates high glucose-induced receptor for advanced glycation end products (RAGE) expression in human embryonic kidney cells.Relationship between downregulation of miRNAs and increase of oxidative stress in the development of diabetic cardiac dysfunction: junctin as a target protein of miR-1.Hydrogen sulfide attenuates the development of diabetic cardiomyopathy.Astragalus polysaccharides protect cardiac stem and progenitor cells by the inhibition of oxidative stress-mediated apoptosis in diabetic hearts.Spironolactone Protects against Diabetic Cardiomyopathy in Streptozotocin-Induced Diabetic Rats Astragalus polysaccharides inhibit oxidation in high glucose-challenged or SOD2-silenced H9C2 cells

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Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

http://www.wikidata.org/entity/Q37951439

description

2010 nî lūn-bûn

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2010年の論文

@ja

2010年学术文章

@wuu

2010年学术文章

@zh-cn

2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

@yue

2010年學術文章

@zh

2010年學術文章

@zh-hant

name

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

@en

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

@nl

type

label

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

@en

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

@nl

prefLabel

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

@en

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

@nl

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Current cardiology reviews

P1476

Role of differential signaling pathways and oxidative stress in diabetic cardiomyopathy.

@en

P2093

Arun Prasath Laksmanan

http://www.w3.org/2001/XMLSchema#string

Flori R Sari

http://www.w3.org/2001/XMLSchema#string

Kenichi Watanabe

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Makoto Kodama

http://www.w3.org/2001/XMLSchema#string

Meilei Harima

http://www.w3.org/2001/XMLSchema#string

Narasimman Gurusamy

http://www.w3.org/2001/XMLSchema#string

Punniyakoti T Veeraveedu

http://www.w3.org/2001/XMLSchema#string

Rajarajan A Thandavarayan

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Sayaka Mito

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Vijayakumar Sukumaran

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P2860

Stress-induced inhibition of ERK1 and ERK2 by direct interaction with p38 MAP kinase

Influence of diabetes mellitus on heart failure risk and outcome

Molecular and cellular mechanisms of cardiotoxicity.

Specificity of receptor tyrosine kinase signaling: transient versus sustained extracellular signal-regulated kinase activation

Apoptosis signal-regulating kinase 1 (ASK1) induces neuronal differentiation and survival of PC12 cells

The mechanism of alloxan and streptozotocin action in B cells of the rat pancreas

Isoproterenol-induced myocardial fibrosis in relation to myocyte necrosis

Activated glycogen synthase-3 beta suppresses cardiac hypertrophy in vivo

p22phox is a critical component of the superoxide-generating NADH/NADPH oxidase system and regulates angiotensin II-induced hypertrophy in vascular smooth muscle cells

A cytoplasmic inhibitor of the JNK signal transduction pathway

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280-290

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10.2174/157340310793566145

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4

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