Osteocytes: central conductors of bone biology in normal and pathological conditions. - Wikidata - wikimedia - TriplyDB

Osteocytes: central conductors of bone biology in normal and pathological conditions.

Osteocytes: central conductors of bone biology in normal and pathological conditions.

http://www.wikidata.org/entity/Q37958204

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Endocrine crosstalk between muscle and bone Bone Cells Dynamics during Peri-Implantitis: a Theoretical Analysis The role of osteocytes in targeted bone remodeling: a mathematical model.The role of polyunsaturated ω-3 fatty acid eicosapentaenoic acid-derived resolvin E1 (RvE1) in bone preservation.Role of FGF/FGFR signaling in skeletal development and homeostasis: learning from mouse models.Impact of extracellular matrix derived from osteoarthritis subchondral bone osteoblasts on osteocytes: role of integrinβ1 and focal adhesion kinase signaling cues.Mechanical Strain Affects Some Microrna Profiles in Pre-Oeteoblasts.UTP-induced ATP release is a fine-tuned signalling pathway in osteocytes.Serum sclerostin is decreased following vitamin D treatment in young vitamin D-deficient female adults.Mechanical forces regulate stem cell response to surface topography.

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Osteocytes: central conductors of bone biology in normal and pathological conditions.

http://www.wikidata.org/entity/Q37958204

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Osteocytes: central conductors of bone biology in normal and pathological conditions.

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Osteocytes: central conductors of bone biology in normal and pathological conditions.

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Osteocytes: central conductors of bone biology in normal and pathological conditions.

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Osteocytes: central conductors of bone biology in normal and pathological conditions.

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Osteocytes: central conductors of bone biology in normal and pathological conditions.

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Osteocytes: central conductors of bone biology in normal and pathological conditions.

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J.1748-1716.2011.02385.X

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Acta Physiologica

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Osteocytes: central conductors of bone biology in normal and pathological conditions.

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P2860

In vivo regulation of apoptosis in metaphyseal trabecular bone of young rats by synthetic human parathyroid hormone (1-34) fragment

Increased bone density in sclerosteosis is due to the deficiency of a novel secreted protein (SOST)

Osteocyte control of bone formation via sclerostin, a novel BMP antagonist

FGF-23 is a potent regulator of vitamin D metabolism and phosphate homeostasis

Connexin 43 (GJA1) mutations cause the pleiotropic phenotype of oculodentodigital dysplasia

Identification of a 52 kb deletion downstream of the SOST gene in patients with van Buchem disease

Control of bone mass and remodeling by PTH receptor signaling in osteocytes

Sclerostin binds to LRP5/6 and antagonizes canonical Wnt signaling

Matrix-embedded cells control osteoclast formation

Evidence for osteocyte regulation of bone homeostasis through RANKL expression

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317-330

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10.1111/J.1748-1716.2011.02385.X

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2012-01-19T00:00:00Z

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