Immunological mechanisms of contact hypersensitivity in mice. - Wikidata - wikimedia - TriplyDB

Immunological mechanisms of contact hypersensitivity in mice.

Immunological mechanisms of contact hypersensitivity in mice.

http://www.wikidata.org/entity/Q37965921

about

Toluene diisocyanate (TDI) disposition and co-localization of immune cells in hair follicles Hapten-induced contact hypersensitivity, autoimmune reactions, and tumor regression: plausibility of mediating antitumor immunity Anti-inflammatory potential of Capparis spinosa L. in vivo in mice through inhibition of cell infiltration and cytokine gene expression Basophils are required for the induction of Th2 immunity to haptens and peptide antigens.Enhanced scratching elicited by a pruritogen and an algogen in a mouse model of contact hypersensitivity.AHAPS-functionalized silica nanoparticles do not modulate allergic contact dermatitis in mice Suppression of antigen-specific adaptive immunity by IL-37 via induction of tolerogenic dendritic cells.The role of neutrophils and G-CSF in DNFB-induced contact hypersensitivity in mice.Sphingosine 1-phosphate modulates antigen capture by murine Langerhans cells via the S1P2 receptor subtype.Afadin requirement for cytokine expressions in keratinocytes during chemically induced inflammation in mice Contact hypersensitivity to oxazolone provokes vulvar mechanical hyperalgesia in mice.Oxazolone-induced contact hypersensitivity reduces lymphatic drainage but enhances the induction of adaptive immunity.The NF-κB regulator Bcl-3 modulates inflammation during contact hypersensitivity reactions in radioresistant cells.CXCR3 chemokine receptor signaling mediates itch in experimental allergic contact dermatitis Common clonal origin of central and resident memory T cells following skin immunization Dermal γδ T Cells Do Not Freely Re-Circulate Out of Skin and Produce IL-17 to Promote Neutrophil Infiltration during Primary Contact Hypersensitivity IL-34 is a tissue-restricted ligand of CSF1R required for the development of Langerhans cells and microglia.Enhanced excitability of MRGPRA3- and MRGPRD-positive nociceptors in a model of inflammatory itch and pain ASK1 promotes the contact hypersensitivity response through IL-17 production.Contact dermatitis: a comparative and translational review of the literature.ELR(+) chemokine-mediated neutrophil recruitment is involved in 2,4,6-trinitrochlorobenzene-induced contact hypersensitivity.Potential applications of keratinocytes derived from human embryonic stem cells.Human embryonic stem cells derived keratinocyte as an in vitro research model for the study of immune response.Construction and phenotypic analysis of mice carrying a duplication of the major histocompatibility class I (MHC-I) locus.Allergic Contact Dermatitis: A Model of Inflammatory Itch and Pain in Human and Mouse.A generally applicable translational strategy identifies S100A4 as a candidate gene in allergy.Aquaporin-9-expressing neutrophils are required for the establishment of contact hypersensitivity.Biological clock dysfunction exacerbates contact hypersensitivity in mice.Topical administration of the pan-Src kinase inhibitors, dasatinib and LCB 03-0110, prevents allergic contact dermatitis in mice.Local and systemic effects of co-stimulatory blockade using cytotoxic T lymphocyte antigen-4-immunoglobulin in dinitrofluorobenzene- and oxazolone-induced contact hypersensitivity in mice.Sensory TRP channels contribute differentially to skin inflammation and persistent itch.The Cooperative Role of CD326+ and CD11b+ Dendritic Cell Subsets for a Hapten-Induced Th2 Differentiation.The indane diastereoisomers, PH2 and PH5: divergence between their effects in delayed-type hypersensitivity models and a model of colitis.Local Hyperthermia Affects Murine Contact Hypersensitivity around Elicitation Phase.Curcumin inhibits epigen and amphiregulin upregulated by 2,4,6-trinitrochlorobenzene associated with attenuation of skin swelling.The balance between pro- and anti-inflammatory cytokines is crucial in human allergic contact dermatitis pathogenesis: the role of IL-1 family members.Anti-inflammatory Effects of Brassica oleracea Var. capitata L. (Cabbage) Methanol Extract in Mice with Contact Dermatitis.Depletion of regulatory T cells in a hapten-induced inflammation model results in prolonged and increased inflammation driven by T cells Mrgprs activation is required for chronic itch conditions in mice Stat5 gene dosage in T cells modulates CD8+T-cell homeostasis and attenuates contact hypersensitivity response in mice

P2860

Q23909608-7F6C82AC-E065-4FEE-A16D-D9CD025FC835 Q28387629-5C551494-285F-4541-8539-F45810FFF93B Q28817812-FEF15170-2C87-404E-ADD6-EE921990C14D Q30539528-932A1A15-AA19-468C-9AF3-9F5F6FB25279 Q34067012-05B2354A-A5A5-45B7-A223-B916DB33A1C2 Q34254990-420A70D5-F0D6-4BC6-825C-DD14E68B846D Q34408962-4C45FDF5-54E1-40A7-B5DF-51BC6BB0F227 Q34454707-1CC308B6-8558-4052-ADBB-F43DFCD2B6AD Q34474499-160A0C78-A53B-4982-A5CF-25DC028FD63E Q34507244-83B90705-4044-4A0F-A959-86CE4ABAD70E Q35036636-A70FA609-8EAA-45C4-883C-D024C4BEBF19 Q35184089-54F6714B-47BF-488E-83A2-4904F34805DE Q35609977-07DC5430-88C1-4C68-8F28-EA91942B76D7 Q35985416-1EF0D1D2-84CC-4B1A-9BF4-3B96F37699C7 Q36246184-12C14CDA-2C87-4B50-8418-8CC43926B372 Q36247489-29F26BB6-E151-4BFC-A27A-066BC3E9BAD4 Q37615661-4B6F7EBA-C203-46F1-86B5-ADFCC7D6E8EB Q37647764-AA0E8312-6B46-4A11-A6FB-F3D1C6E68EBF Q37705602-C612CE69-3EAC-44B8-BEA4-5E55572B3E55 Q38549107-75388695-EDA1-4F3B-98CF-EBB878B27755 Q38649819-8076723F-D54F-4D67-86F8-AFD078ABFDEA Q38668504-93771D2A-0EE9-4DE1-83F0-F20139E76D1D Q39184806-09813976-2ADB-48E0-8F2D-F34A73099319 Q39294873-1E922977-1D05-4767-9B7B-179070A48E85 Q41811724-948C9BC5-2E51-40BA-AB14-F3FA901D1D9C Q42112839-68588682-5334-4B11-BF66-F5BC5D31F455 Q43207291-EC52055E-CFC2-4BAD-946A-26B802FF6783 Q44544254-5F40DFF0-ED16-493D-86E5-4599188DF1BF Q45764606-FABD391F-45EA-4AF5-B605-86760E9C2D1B Q46614633-3E968632-ED4B-4F2A-8BF4-9543061E5EF7 Q47132169-0E27D981-0DB8-488F-A63B-387E8814E8AA Q47709675-5B24C913-6224-465C-B14A-7ADF45F928C2 Q47745802-CCF4C678-94F3-46BB-B84E-DA6566CCAA9C Q49294273-1CBDD996-94AD-486D-BF2C-00FA8CF2FC1B Q52777064-FDFFDE88-0651-4AB8-B60A-E2317762663C Q54688502-65CCD595-F52D-4A3F-B035-CDECBFFB080A Q55317497-71E43173-0B25-4D7B-8B8A-B5680F5789DE Q56896192-AA39B08D-7BF6-45D9-A659-142732E3B254 Q57134299-902008F3-EB27-4799-AF82-3B29EE03CE6B Q58455445-E869EBB2-7F7B-42C2-8EF7-C7B5CD7F19B1

P2860

Immunological mechanisms of contact hypersensitivity in mice.

http://www.wikidata.org/entity/Q37965921

description

2011 nî lūn-bûn

@nan

2011年の論文

@ja

2011年学术文章

@wuu

2011年学术文章

@zh-cn

2011年学术文章

@zh-hans

2011年学术文章

@zh-my

2011年学术文章

@zh-sg

2011年學術文章

@yue

2011年學術文章

@zh

2011年學術文章

@zh-hant

name

Immunological mechanisms of contact hypersensitivity in mice.

@en

Immunological mechanisms of contact hypersensitivity in mice.

@nl

type

label

Immunological mechanisms of contact hypersensitivity in mice.

@en

Immunological mechanisms of contact hypersensitivity in mice.

@nl

prefLabel

Immunological mechanisms of contact hypersensitivity in mice.

@en

Immunological mechanisms of contact hypersensitivity in mice.

@nl

P1433

Q37965921-4508643E-A329-4BA0-8619-34A40842D7C8

P1476

Q37965921-84ECFC02-88B2-486A-B713-9E697547B5FB

P2093

Q37965921-00DD4982-7D22-4B91-BA2B-D5205421F95D

Q37965921-A4F016A7-A114-44A4-A5B2-90DF147E659F

P2860

Q37965921-011F88A7-58C3-45C4-A159-AFC115EBAD09

Q37965921-04408B76-8F2A-4335-A607-A14C421AFFE1

Q37965921-097B3355-2AA6-4C8B-A86B-D5527E599EAE

Q37965921-0B696C92-9F15-4EBA-96A6-0D317251F074

Q37965921-0ED2A055-B7C5-493D-850D-B51AD0A8FB2F

Q37965921-179700D1-F78F-4DD3-A3BC-42CCE2F8C2E3

Q37965921-19DDCAE0-6E09-4D1F-98EC-A6BB1ACB08B2

Q37965921-1BB8AEEA-3F1F-499D-9842-1BC61E6736EB

Q37965921-1C6725E6-8957-498C-95A3-730F395D64A0

Q37965921-1DBA5833-7127-4BCF-93C4-48126D2DFEDD

P304

Q37965921-4729E5C8-E9B5-4DD3-B968-E25584901FF9

P31

Q37965921-44B6460D-F76F-416F-8401-B4ABF2711390

P356

Q37965921-C71B2F29-C208-405E-80AE-10E4AA4B618C

P433

Q37965921-1A7465D5-3CB4-49DB-938B-4E8F37685EA1

P478

Q37965921-129B0207-A521-48BF-9E82-3E2BA1D25145

P577

Q37965921-20CD2452-52B7-424E-8F06-8BBEFD728070

P5875

Q37965921-54E875CF-F9E6-4B57-832F-DAC370853821

P698

Q37965921-93FAD9FA-5C38-4137-8EAE-B7D4BAECA8A6

P921

Q37965921-46F2A53B-BE27-4861-AD0B-E4405090DB65

P356

J.1600-0463.2011.02832.X

P1433

Acta Pathologica et Microbiologica Scandinavica

P1476

Immunological mechanisms of contact hypersensitivity in mice.

@en

P2093

Anne Deen Christensen

http://www.w3.org/2001/XMLSchema#string

Claus Haase

http://www.w3.org/2001/XMLSchema#string

P2860

Processing of urushiol (poison ivy) hapten by both endogenous and exogenous pathways for presentation to T cells in vitro

Mechanism of dinitrochlorobenzene-induced dermatitis in mice: role of specific antibodies in pathogenesis

Interleukin-21 inhibits dendritic cell activation and maturation

Three-dimensional segregation of supramolecular activation clusters in T cells

T cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine production

Expression and function of B7-1 and B7-2 in hapten-induced contact sensitivity

IL-20 receptor 2 signaling down-regulates antigen-specific T cell responses

CCR7 coordinates the primary immune response by establishing functional microenvironments in secondary lymphoid organs

Identification of DC-SIGN, a novel dendritic cell-specific ICAM-3 receptor that supports primary immune responses

Visualizing CD4 T-cell migration into inflamed skin and its inhibition by CCR4/CCR10 blockades using in vivo imaging model.

P304

1-27

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1111/J.1600-0463.2011.02832.X

http://www.w3.org/2001/XMLSchema#string

P433

1

http://www.w3.org/2001/XMLSchema#string

P478

120

http://www.w3.org/2001/XMLSchema#string

P577

2011-11-11T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

51861600

http://www.w3.org/2001/XMLSchema#string

P698

22151305

http://www.w3.org/2001/XMLSchema#string

P921