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Multiple sclerosis: molecular mechanisms and therapeutic opportunitiesControl of autoimmune CNS inflammation by astrocytesCentral SDF-1/CXCL12 expression and its cardiovascular and sympathetic effects: the role of angiotensin II, TNF-α, and MAP kinase signaling.Prenatal stress is a vulnerability factor for altered morphology and biological activity of microglia cellsUtility of CSF Cytokine/Chemokines as Markers of Active Intrathecal Inflammation: Comparison of Demyelinating, Anti-NMDAR and Enteroviral EncephalitisCorrelation of Gut Microbiota Composition with Resistance to Experimental Autoimmune Encephalomyelitis in Rats.The peculiarities of the SDF-1/CXCL12 system: in some cells, CXCR4 and CXCR7 sing solos, in others, they sing duets.Betulinic acid regulates generation of neuroinflammatory mediators responsible for tissue destruction in multiple sclerosis in vitro.Nitric oxide inhibits CXCL12 expression in neuroinflammation.Gut Microbiota Confers Resistance of Albino Oxford Rats to the Induction of Experimental Autoimmune Encephalomyelitis.
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P2860
description
article científic
@ca
article scientifique
@fr
articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
@pt
artigo científico
@pt-br
artikel ilmiah
@id
artikull shkencor
@sq
artículo científico
@es
name
CXCL12 in control of neuroinflammation.
@en
CXCL12 in control of neuroinflammation.
@nl
type
label
CXCL12 in control of neuroinflammation.
@en
CXCL12 in control of neuroinflammation.
@nl
prefLabel
CXCL12 in control of neuroinflammation.
@en
CXCL12 in control of neuroinflammation.
@nl
P2860
P1433
P1476
CXCL12 in control of neuroinflammation
@en
P2093
Marija Mostarica-Stojković
P2860
P2888
P356
10.1007/S12026-012-8282-X
P577
2012-04-01T00:00:00Z