The stroke-prone spontaneously hypertensive rat: still a useful model for post-GWAS genetic studies?
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Mitochondrial Dysfunction Contributes to Hypertensive Target Organ Damage: Lessons from an Animal Model of Human DiseaseGenetic, physiological and comparative genomic studies of hypertension and insulin resistance in the spontaneously hypertensive ratDifferential gene expression in multiple neurological, inflammatory and connective tissue pathways in a spontaneous model of human small vessel stroke.Towards Precision Medicine for Hypertension: A Review of Genomic, Epigenomic, and Microbiomic Effects on Blood Pressure in Experimental Rat Models and Humans.Loss of FMRP Impaired Hippocampal Long-Term Plasticity and Spatial Learning in Rats.Loss of FMRP Impaired Hippocampal Long-Term Plasticity and Spatial Learning in Rats.Mycophenolate mofetil prevents cerebrovascular injury in stroke-prone spontaneously hypertensive rats.Utilizing proteomics to understand and define hypertension: where are we and where do we go?
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The stroke-prone spontaneously hypertensive rat: still a useful model for post-GWAS genetic studies?
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The stroke-prone spontaneously ...... for post-GWAS genetic studies?
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The stroke-prone spontaneously ...... for post-GWAS genetic studies?
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The stroke-prone spontaneously ...... for post-GWAS genetic studies?
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The stroke-prone spontaneously ...... for post-GWAS genetic studies?
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The stroke-prone spontaneously ...... for post-GWAS genetic studies?
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The stroke-prone spontaneously ...... for post-GWAS genetic studies?
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P2093
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The stroke-prone spontaneously ...... for post-GWAS genetic studies?
@en
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Hiroki Ohara
Minoru Isomura
Norihiro Kato
Toru Nabika
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10.1038/HR.2012.30
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2012-03-08T00:00:00Z