Revisiting the old link between infection and autoimmune disease with commensals and T helper 17 cells. - Wikidata - wikimedia - TriplyDB

Revisiting the old link between infection and autoimmune disease with commensals and T helper 17 cells.

Revisiting the old link between infection and autoimmune disease with commensals and T helper 17 cells.

http://www.wikidata.org/entity/Q37998396

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Microbial view of central nervous system autoimmunity The Autoimmune Ecology Role of viruses and bacteria-virus interactions in autoimmunity Death-defining immune responses after apoptosis The MHC locus and genetic susceptibility to autoimmune and infectious diseases.Coherent somatic mutation in autoimmune disease.Inflammation and vitamin D: the infection connection Unresolved issues in theories of autoimmune disease using myocarditis as a framework Apoptosis in response to microbial infection induces autoreactive TH17 cells Complexities in the relationship between infection and autoimmunity.Close Encounters of the First Kind: Innate Sensors and Multiple Sclerosis.The autoimmunity-oral microbiome connection.Regulation of inflammation by microbiota interactions with the host.Rheumatic diseases and the microbiome

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Revisiting the old link between infection and autoimmune disease with commensals and T helper 17 cells.

http://www.wikidata.org/entity/Q37998396

description

article científic

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article scientifique

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articolo scientifico

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artikel ilmiah

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artículo científico

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Revisiting the old link betwee ...... mensals and T helper 17 cells.

@en

Revisiting the old link betwee ...... mensals and T helper 17 cells.

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type

label

Revisiting the old link betwee ...... mensals and T helper 17 cells.

@en

Revisiting the old link betwee ...... mensals and T helper 17 cells.

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prefLabel

Revisiting the old link betwee ...... mensals and T helper 17 cells.

@en

Revisiting the old link betwee ...... mensals and T helper 17 cells.

@nl

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S12026-012-8311-9

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Immunologic Research

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Revisiting the old link betwee ...... mensals and T helper 17 cells.

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J Magarian Blander

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Laura Campisi

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Miriam B Torchinsky

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P2860

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Environmental risk factors for multiple sclerosis. Part I: The role of infection

The inflammasomes

IL-23 is increased in dendritic cells in multiple sclerosis and down-regulation of IL-23 by antisense oligos increases dendritic cell IL-10 production

Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls

The interleukin 23 receptor is essential for the terminal differentiation of interleukin 17-producing effector T helper cells in vivo

Virus-plus-susceptibility gene interaction determines Crohn's disease gene Atg16L1 phenotypes in intestine

T helper 17 cell heterogeneity and pathogenicity in autoimmune disease

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s12026-012-8311-9

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50-68

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scholarly article

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10.1007/S12026-012-8311-9

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1-3

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54

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2012-12-01T00:00:00Z

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22460741

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